Found: 12
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Protein carbonylation: Proteomics, specificity and relevance to aging.
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- Mass Spectrometry Reviews, 2014, v. 33, n. 1, p. 21, doi. 10.1002/mas.21375
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- Article
Apoptotic cell death and altered calcium homeostasis caused by frataxin depletion in dorsal root ganglia neurons can be prevented by BH4 domain of Bcl-xL protein.
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- Human Molecular Genetics, 2014, v. 23, n. 7, p. 1829, doi. 10.1093/hmg/ddt576
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- Article
Evolution of an Iron-Detoxifying Protein: Eukaryotic and Rickettsia Frataxins Contain a Conserved Site Which Is Not Present in Their Bacterial Homologues.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 21, p. 13151, doi. 10.3390/ijms232113151
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- Article
Calcitriol increases frataxin levels and restores mitochondrial function in cell models of Friedreich Ataxia.
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- Biochemical Journal, 2021, v. 478, n. 1, p. 1, doi. 10.1042/BCJ20200331
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- Article
Calpain-Inhibitors Protect Frataxin-Deficient Dorsal Root Ganglia Neurons from Loss of Mitochondrial Na+/Ca2+ Exchanger, NCLX, and Apoptosis.
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- Neurochemical Research, 2021, v. 46, n. 1, p. 108, doi. 10.1007/s11064-020-03020-3
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- Article
Mitochondrial iron and calcium homeostasis in Friedreich ataxia.
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- IUBMB Life, 2021, v. 73, n. 3, p. 543, doi. 10.1002/iub.2457
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- Article
Protein oxidation in Huntington disease.
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- Biofactors, 2012, v. 38, n. 3, p. 173, doi. 10.1002/biof.1013
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- Article
Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.
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- Cellular & Molecular Life Sciences, 2024, v. 81, n. 1, p. 1, doi. 10.1007/s00018-023-05064-4
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- Article
Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.
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- Cellular & Molecular Life Sciences, 2022, v. 79, n. 2, p. 1, doi. 10.1007/s00018-021-04100-5
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- Article
Calcitriol Treatment Is Safe and Increases Frataxin Levels in Friedreich Ataxia Patients.
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- Movement Disorders, 2024, v. 39, n. 7, p. 1099, doi. 10.1002/mds.29808
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- Article
Frataxin‐deficient neurons and mice models of Friedreich ataxia are improved by TAT‐MTScs‐FXN treatment.
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- Journal of Cellular & Molecular Medicine, 2018, v. 22, n. 2, p. 834, doi. 10.1111/jcmm.13365
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- Article
Iron in Friedreich Ataxia: A Central Role in the Pathophysiology or an Epiphenomenon?
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- Pharmaceuticals (14248247), 2018, v. 11, n. 3, p. 89, doi. 10.3390/ph11030089
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- Article