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IL-1α and IL-36 Family Cytokines Can Undergo Processing and Activation by Diverse Allergen-Associated Proteases.
- Published in:
- Frontiers in Immunology, 2022, v. 13, p. 1, doi. 10.3389/fimmu.2022.879029
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- Article
Suppressing IL-36-driven inflammation using peptide pseudosubstrates for neutrophil proteases.
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- Cell Death & Disease, 2018, v. 9, n. 3, p. 1, doi. 10.1038/s41419-018-0385-4
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- Article
Identification of small‐molecule elastase inhibitors as antagonists of IL‐36 cytokine activation.
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- FEBS Open Bio, 2018, v. 8, n. 5, p. 751, doi. 10.1002/2211-5463.12406
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- Article
Myeloid cell–derived proteases produce a proinflammatory form of IL-37 that signals via IL-36 receptor engagement.
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- Science Immunology, 2022, v. 7, n. 78, p. 1, doi. 10.1126/sciimmunol.ade5728
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- Article
The path to venetoclax resistance is paved with mutations, metabolism, and more.
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- Science Translational Medicine, 2022, v. 14, n. 674, p. 1, doi. 10.1126/scitranslmed.abo6891
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- Article
Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokines.
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- FEBS Journal, 2017, v. 284, n. 11, p. 1712, doi. 10.1111/febs.14075
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- Article