Found: 20
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Rhes Deletion Is Neuroprotective in the 3-Nitropropionic Acid Model of Huntington's Disease.
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- Journal of Neuroscience, 2013, v. 33, n. 9, p. 4206, doi. 10.1523/JNEUROSCI.3730-12.2013
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- Article
Global Rhes knockout in the Q175 Huntington's disease mouse model.
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- PLoS ONE, 2021, v. 16, n. 10, p. 1, doi. 10.1371/journal.pone.0258486
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- Article
Elevated P75<sup>NTR</sup> expression causes death of engrailed-deficient midbrain dopaminergic neurons by Erk1/2 suppression.
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- Neural Development, 2009, v. 4, p. 1, doi. 10.1186/1749-8104-4-11
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- Article
Exaggerated mitophagy: a weapon of striatal destruction in the brain?
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- Biochemical Society Transactions, 2020, v. 48, n. 2, p. 709, doi. 10.1042/BST20191283
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- Article
GDNF prevents TGF-β-induced damage of the plasma membrane in cerebellar granule neurons by suppressing activation of p38-MAPK via the phosphatidylinositol 3-kinase pathway.
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- Cell & Tissue Research, 2008, v. 331, n. 2, p. 373, doi. 10.1007/s00441-007-0538-8
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- Article
Mutant Huntingtin stalls ribosomes and represses protein synthesis in a cellular model of Huntington disease.
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- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-21637-y
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- Article
Abnormal RasGRP1 Expression in the Post-Mortem Brain and Blood Serum of Schizophrenia Patients.
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- Biomolecules (2218-273X), 2022, v. 12, n. 2, p. 328, doi. 10.3390/biom12020328
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- Article
Tunneling Nanotube: An Enticing Cell–Cell Communication in the Nervous System.
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- Biology (2079-7737), 2023, v. 12, n. 10, p. 1288, doi. 10.3390/biology12101288
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- Article
Rhes, a striatal enriched protein, regulates post-translational small-ubiquitin-like-modifier (SUMO) modification of nuclear proteins and alters gene expression.
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- Cellular & Molecular Life Sciences, 2024, v. 81, n. 1, p. 1, doi. 10.1007/s00018-024-05181-8
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- Article
TGF-β induces cell death in the oligodendroglial cell line OLI-neu.
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- Glia, 2002, v. 40, n. 1, p. 95, doi. 10.1002/glia.10110
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- Article
RasGRP1 promotes amphetamine-induced motor behavior through a Rhes interaction network ("Rhesactome") in the striatum.
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- Science Signaling, 2016, v. 9, n. 454, p. 1, doi. 10.1126/scisignal.aaf6670
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- Article
Hunting tin promotes mTORC1 signaling in the pathogenesis of Huntington's disease.
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- Science Signaling, 2014, v. 7, n. 349, p. 1, doi. 10.1126/scisignal.2005633
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- Article
Tau Aggregation and Progressive Neuronal Degeneration in the Absence of Changes in Spine Density and Morphology after Targeted Expression of Alzheimer's Disease-Relevant Tau Constructs in Organotypic Hippocampal Slices.
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- Journal of Neuroscience, 2006, v. 26, n. 22, p. 6103, doi. 10.1523/JNEUROSCI.4245-05.2006
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- Article
Insulin-Like Growth Factor 1 Inhibits Extracellular Signal-Regulated Kinase to Promote Neuronal Survival via the Phosphatidylinositol 3-Kinase/Protein Kinase A/c-Raf Pathway.
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- Journal of Neuroscience, 2005, v. 25, n. 11, p. 2838, doi. 10.1523/JNEUROSCI.5060-04.2005
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- Article
Glutamate activates NF-κB through calpain in neurons.
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- European Journal of Neuroscience, 2003, v. 18, n. 12, p. 3305, doi. 10.1111/j.1460-9568.2003.03079.x
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- Article
Role of Transient Receptor Potential Channel 1 (TRPC1) in Glutamate-Induced Cell Death in the Hippocampal Cell Line HT22.
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- Journal of Molecular Neuroscience, 2014, v. 52, n. 3, p. 425, doi. 10.1007/s12031-013-0171-9
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- Article
Rhes, a striatal-enriched small G protein, mediates mTOR signaling and L-DOPA-induced dyskinesia.
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- Nature Neuroscience, 2012, v. 15, n. 2, p. 191, doi. 10.1038/nn.2994
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- Article
The mammalian target of rapamycin (mTOR) kinase mediates haloperidol-induced cataleptic behavior.
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- Translational Psychiatry, 2020, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41398-020-01014-x
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- Article
Rhes Tunnels: A Radical New Way of Communication in the Brain's Striatum?
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- BioEssays, 2020, v. 42, n. 6, p. 1, doi. 10.1002/bies.201900231
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ERK and cell death: ERK1/2 in neuronal death.
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- FEBS Journal, 2010, v. 277, n. 1, p. 22, doi. 10.1111/j.1742-4658.2009.07367.x
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- Article