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TDP-43 regulates LC3ylation in neural tissue through ATG4B cryptic splicing inhibition.
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- Acta Neuropathologica, 2024, v. 148, n. 1, p. 1, doi. 10.1007/s00401-024-02780-4
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- Article
NF-κB Signaling Pathways: Role in Nervous System Physiology and Pathology.
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- Neuroscientist, 2013, v. 19, n. 2, p. 175, doi. 10.1177/1073858412444007
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- Article
Calpain system is altered in survival motor neuron-reduced cells from in vitro and in vivo spinal muscular atrophy models.
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- Cell Death & Disease, 2020, v. 11, n. 6, p. 1, doi. 10.1038/s41419-020-2688-5
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- Article
Inhibition of autophagy delays motoneuron degeneration and extends lifespan in a mouse model of spinal muscular atrophy.
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- Cell Death & Disease, 2017, v. 8, n. 12, p. 1, doi. 10.1038/s41419-017-0086-4
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- Article
Reply.
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- Acta Neurologica Scandinavica, 1994, v. 89, n. 3, p. 235, doi. 10.1111/j.1600-0404.1994.tb01671.x
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- Article
Reply.
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- Acta Neurologica Scandinavica, 1992, v. 86, n. 4, p. 428, doi. 10.1111/j.1600-0404.1992.tb05114.x
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- Article
The Canonical Nuclear Factor-κB Pathway Regulates Cell Survival in a Developmental Model of Spinal Cord Motoneurons.
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- Journal of Neuroscience, 2011, v. 31, n. 17, p. 6493, doi. 10.1523/JNEUROSCI.0206-11.2011
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- Article
The Death Receptor Antagonist FLIP-L Interacts with Trk and Is Necessary for Neurite Outgrowth Induced by Neurotrophins.
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- Journal of Neuroscience, 2010, v. 30, n. 17, p. 6094, doi. 10.1523/JNEUROSCI.0537-10.2010
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- Article
Specific vulnerability of mouse spinal cord motoneurons to membrane depolarization.
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- Journal of Neurochemistry, 2009, v. 110, n. 6, p. 1842, doi. 10.1111/j.1471-4159.2009.06278.x
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- Article
Tyr-701 is a new regulatory site for neurotrophin receptor TrkA trafficking and function.
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- Journal of Neurochemistry, 2008, v. 104, n. 1, p. 124, doi. 10.1111/j.1471-4159.2007.05027.x
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- Article
ERK MAPK signaling pathway inhibition as a potential target to prevent autophagy alterations in Spinal Muscular Atrophy motoneurons.
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- Cell Death Discovery, 2023, v. 9, n. 1, p. 1, doi. 10.1038/s41420-023-01409-x
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- Article
Notch Signaling Pathway Is Activated in Motoneurons of Spinal Muscular Atrophy.
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- International Journal of Molecular Sciences, 2013, v. 14, n. 6, p. 11424, doi. 10.3390/ijms140611424
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- Article
Calpain Inhibition Increases SMN Protein in Spinal Cord Motoneurons and Ameliorates the Spinal Muscular Atrophy Phenotype in Mice.
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- Molecular Neurobiology, 2019, v. 56, n. 6, p. 4414, doi. 10.1007/s12035-018-1379-z
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- Article
Regulation of Survival Motor Neuron Protein by the Nuclear Factor-Kappa B Pathway in Mouse Spinal Cord Motoneurons.
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- Molecular Neurobiology, 2018, v. 55, n. 6, p. 5019, doi. 10.1007/s12035-017-0710-4
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- Publication type:
- Article
Spinal Muscular Atrophy autophagy profile is tissue-dependent: differential regulation between muscle and motoneurons.
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- Acta Neuropathologica Communications, 2021, v. 9, n. 1, p. 1, doi. 10.1186/s40478-021-01223-5
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- Article
Activation of Phosphatidylinositol 3-Kinase, but Not Extracellular-Regulated Kinases, Is Necessary to Mediate Brain-Derived Neurotrophic Factor-Induced Motoneuron Survival.
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- Journal of Neurochemistry, 1999, v. 73, n. 2, p. 521, doi. 10.1046/j.1471-4159.1999.0730521.x
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- Article
Survival motor neuron protein and neurite degeneration are regulated by Gemin3 in spinal muscular atrophy motoneurons.
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- Frontiers in Cellular Neuroscience, 2022, v. 16, p. 1, doi. 10.3389/fncel.2022.1054270
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- Article
Sp1-regulated expression of p11 contributes to motor neuron degeneration by membrane insertion of TASK1.
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- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-11637-4
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- Article
Smn-Deficiency Increases the Intrinsic Excitability of Motoneurons.
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- Frontiers in Cellular Neuroscience, 2017, p. 1, doi. 10.3389/fncel.2017.00269
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- Article
Neuronal survival induced by neurotrophins requires calmodulin.
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- Journal of Cell Biology, 2001, v. 154, n. 3, p. 585, doi. 10.1083/jcb.200101023
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- Article
The Y172 Monoclonal Antibody Against p-c-Jun (Ser63) Is a Marker of the Postsynaptic Compartment of C-Type Cholinergic Afferent Synapses on Motoneurons.
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- Frontiers in Cellular Neuroscience, 2020, v. 13, p. 1, doi. 10.3389/fncel.2019.00582
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- Article