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Ncx3-Induced Mitochondrial Dysfunction in Midbrain Leads to Neuroinflammation in Striatum of A53t-α-Synuclein Transgenic Old Mice.
- Published in:
- International Journal of Molecular Sciences, 2021, v. 22, n. 15, p. 8177, doi. 10.3390/ijms22158177
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- Article
Expression and function of Kv7.4 channels in rat cardiac mitochondria: possible targets for cardioprotection.
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- Cardiovascular Research, 2016, v. 110, n. 1, p. 40, doi. 10.1093/cvr/cvv281
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- Article
BIOLOGICAL ASPECTS OF NEW MOLECULAR THERAPIES FOR NEUROPATHOLOGIES.
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- Medical Science Pulse, 2024, v. 18, p. 39
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- Article
EXPLORING THE NEUROPROTECTIVE EFFECTS OF SYNTHETIC COMPOUNDS FOR NEW NEURODRUG DEVELOPMENT.
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- 2024
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- Publication type:
- Abstract
Oxidative Metabolism in Brain Ischemia and Preconditioning: Two Sides of the Same Coin.
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- Antioxidants, 2024, v. 13, n. 5, p. 547, doi. 10.3390/antiox13050547
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- Article
NCX1 and NCX3 as potential factors contributing to neurodegeneration and neuroinflammation in the A53T transgenic mouse model of Parkinson’s Disease.
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- Cell Death & Disease, 2018, v. 9, n. 7, p. 1, doi. 10.1038/s41419-018-0775-7
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- Article
L-Ornithine L-Aspartate Restores Mitochondrial Function and Modulates Intracellular Calcium Homeostasis in Parkinson's Disease Models.
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- Cells (2073-4409), 2022, v. 11, n. 18, p. N.PAG, doi. 10.3390/cells11182909
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- Article
Editorial: Advances in brain disorders: from mechanisms to therapeutic targets.
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- Frontiers in Molecular Neuroscience, 2023, p. 1, doi. 10.3389/fnmol.2023.1337583
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- Article
Novel cellular mechanisms for neuroprotection in ischemic preconditioning: a view from inside organelles.
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- Frontiers in Neurology, 2015, v. 6, p. 1, doi. 10.3389/fneur.2015.00115
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- Article
Age-Related Changes in D-Aspartate Oxidase Promoter Methylation Control Extracellular D-Aspartate Levels and Prevent Precocious Cell Death during Brain Aging.
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- Journal of Neuroscience, 2016, v. 36, n. 10, p. 3064, doi. 10.1523/JNEUROSCI.3881-15.2016
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- Article
Knocking-out the Siah2 E3 ubiquitin ligase prevents mitochondrial NCX3 degradation, regulates mitochondrial fission and fusion, and restores mitochondrial function in hypoxic neurons.
- Published in:
- Cell Communication & Signaling, 2020, v. 18, n. 1, p. 1, doi. 10.1186/s12964-020-0529-x
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- Article