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The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury.
- Published in:
- Cell Death & Disease, 2023, v. 14, n. 9, p. 1, doi. 10.1038/s41419-023-06147-7
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- Article
Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair.
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- Cell Death & Disease, 2022, v. 13, n. 8, p. 1, doi. 10.1038/s41419-022-05196-8
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- Article
Arginase 1 promotes retinal neurovascular protection from ischemia through suppression of macrophage inflammatory responses.
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- Cell Death & Disease, 2018, v. 9, n. 10, p. 1, doi. 10.1038/s41419-018-1051-6
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- Article
Arginase 2 promotes neurovascular degeneration during ischemia/reperfusion injury.
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- Cell Death & Disease, 2016, v. 7, n. 11, p. e2483, doi. 10.1038/cddis.2016.295
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- Article
Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury.
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- Journal of Neuroinflammation, 2024, v. 21, n. 1, p. 1, doi. 10.1186/s12974-024-03159-8
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- Article
Is the Arginase Pathway a Novel Therapeutic Avenue for Diabetic Retinopathy?
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- Journal of Clinical Medicine, 2020, v. 9, n. 2, p. 425, doi. 10.3390/jcm9020425
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- Article
Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions.
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- Journal of Neuroinflammation, 2024, v. 21, n. 1, p. 1, doi. 10.1186/s12974-024-03058-y
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- Article
Mechanisms of Diabetes-Induced Endothelial Cell Senescence: Role of Arginase 1.
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- International Journal of Molecular Sciences, 2018, v. 19, n. 4, p. 1215, doi. 10.3390/ijms19041215
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- Article