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The Epstein-Barr virus and the pathogenesis of lymphoma.
- Published in:
- Journal of Pathology, 2015, v. 235, n. 2, p. 312, doi. 10.1002/path.4459
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- Article
MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs.
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- eLife, 2016, p. 1, doi. 10.7554/eLife.18270
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- Article
Pumilio directs deadenylation-associated translational repression of the cyclin-dependent kinase 1 activator RGC-32.
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- Nucleic Acids Research, 2018, v. 46, n. 7, p. 3707, doi. 10.1093/nar/gky038
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- Article
NF-κB directly mediates epigenetic deregulation of common microRNAs in Epstein-Barr virus-mediated transformation of B-cells and in lymphomas.
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- Nucleic Acids Research, 2014, v. 42, n. 17, p. 11025, doi. 10.1093/nar/gku826
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- Article
Epstein--Barr virus-mediated transformation of B cells induces global chromatin changes independent to the acquisition of proliferation.
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- Nucleic Acids Research, 2014, v. 42, n. 1, p. 249, doi. 10.1093/nar/gkt886
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- Article
Differentiation of Human Cytomegalovirus Genotypes in Immunocompromised Patients on the Basis of UL4 Gene Polymorphisms.
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- Journal of Infectious Diseases, 2001, v. 183, n. 2, p. 218, doi. 10.1086/317939
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- Article
The Potential for EBV Vaccines to Prevent Multiple Sclerosis.
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- Frontiers in Neurology, 2022, v. 13, p. 1, doi. 10.3389/fneur.2022.887794
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- Article
Ephrin receptor A2, the epithelial receptor for Epstein-Barr virus entry, is not available for efficient infection in human gastric organoids.
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- PLoS Pathogens, 2021, p. 1, doi. 10.1371/journal.ppat.1009210
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- Article
Deciphering the roles of myeloid derived suppressor cells in viral oncogenesis.
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- Frontiers in Immunology, 2023, v. 14, p. 1, doi. 10.3389/fimmu.2023.1161848
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- Article
The effects of maribavir on the autophosphorylation of ganciclovir resistant mutants of the cytomegalovirus UL97 protein.
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- Herpesviridae, 2010, v. 1, n. 1, p. 1, doi. 10.1186/2042-4280-1-4
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- Article
SARS-CoV-2 Causes a Different Cytokine Response Compared to Other Cytokine Storm-Causing Respiratory Viruses in Severely Ill Patients.
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- Frontiers in Immunology, 2021, v. 11, p. N.PAG, doi. 10.3389/fimmu.2021.629193
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- Article
Epstein-Barr virus B95.8 produced in 293 cells shows marked tropism for differentiated primary epithelial cells and reveals interindividual variation in susceptibility to viral infection.
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- International Journal of Cancer, 2007, v. 121, n. 3, p. 588, doi. 10.1002/ijc.22727
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- Article
The B cell transcription program mediates hypomethylation and overexpression of key genes in Epstein-Barr virus-associated proliferative conversion.
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- Genome Biology, 2013, v. 14, n. 1, p. 1, doi. 10.1186/gb-2013-14-1-r3
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- Article
Early T Cell Recognition of B Cells following Epstein-Barr Virus Infection: Identifying Potential Targets for Prophylactic Vaccination.
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- PLoS Pathogens, 2016, v. 12, n. 4, p. 1, doi. 10.1371/journal.ppat.1005549
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- Article
Cooperation between Epstein-Barr Virus Immune Evasion Proteins Spreads Protection from CD8<sup>+</sup> T Cell Recognition across All Three Phases of the Lytic Cycle.
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- PLoS Pathogens, 2014, v. 10, n. 8, p. 1, doi. 10.1371/journal.ppat.1004322
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- Article
Epstein-Barr Virus Infection of Naïve B Cells In Vitro Frequently Selects Clones with Mutated Immunoglobulin Genotypes: Implications for Virus Biology.
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- PLoS Pathogens, 2012, v. 8, n. 5, p. 1, doi. 10.1371/journal.ppat.1002697
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Epstein-Barr Virus Infection of Polarized Epithelial Cells via the Basolateral Surface by Memory B Cell-Mediated Transfer Infection.
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- PLoS Pathogens, 2011, v. 7, n. 5, p. 1, doi. 10.1371/journal.ppat.1001338
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Stage-Specific Inhibition of MHC Class I Presentation by the Epstein-Barr Virus BNLF2a Protein during Virus Lytic Cycle.
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- PLoS Pathogens, 2009, v. 5, n. 6, p. 1, doi. 10.1371/journal.ppat.1000490
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The Epstein-Barr Virus G-Protein-Coupled Receptor Contributes to Immune Evasion by Targeting MHC Class I Molecules for Degradation.
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- PLoS Pathogens, 2009, v. 5, n. 1, p. 1, doi. 10.1371/journal.ppat.1000255
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The Global Landscape of EBV-Associated Tumors.
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- Frontiers in Oncology, 2019, p. 1, doi. 10.3389/fonc.2019.00713
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- Article