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Correction: Extended Synaptotagmin (ESyt) Triple Knock-Out Mice Are Viable and Fertile without Obvious Endoplasmic Reticulum Dysfunction.
- Published in:
- 2024
- By:
- Publication type:
- Correction Notice
Combinatorial expression of neurexins and LAR-type phosphotyrosine phosphatase receptors instructs assembly of a cerebellar circuit.
- Published in:
- Nature Communications, 2023, v. 14, n. 1, p. 1, doi. 10.1038/s41467-023-40526-0
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- Publication type:
- Article
Deletion of Calsyntenin-3, an atypical cadherin, suppresses inhibitory synapses but increases excitatory parallel-fiber synapses in cerebellum.
- Published in:
- eLife, 2022, p. 1, doi. 10.7554/eLife.70664
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- Publication type:
- Article
Neurexins regulate presynaptic GABAB-receptors at central synapses.
- Published in:
- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-22753-5
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- Publication type:
- Article
Neurexins cluster Ca<sup>2+</sup> channels within the presynaptic active zone.
- Published in:
- EMBO Journal, 2020, v. 39, n. 7, p. 1, doi. 10.15252/embj.2019103208
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- Publication type:
- Article
LAR receptor phospho-tyrosine phosphatases regulate NMDA-receptor responses.
- Published in:
- eLife, 2020, p. 1, doi. 10.7554/eLife.53406
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- Publication type:
- Article
RIM‐binding proteins recruit BK‐channels to presynaptic release sites adjacent to voltage‐gated Ca<sup>2+</sup>‐channels.
- Published in:
- EMBO Journal, 2018, v. 37, n. 16, p. 1, doi. 10.15252/embj.201798637
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- Publication type:
- Article
Evidence of Presynaptic Localization and Function of the c-Jun N-Terminal Kinase.
- Published in:
- Neural Plasticity, 2017, p. 1, doi. 10.1155/2017/6468356
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- Publication type:
- Article
Extended Synaptotagmin (ESyt) Triple Knock-Out Mice Are Viable and Fertile without Obvious Endoplasmic Reticulum Dysfunction.
- Published in:
- PLoS ONE, 2016, v. 11, n. 6, p. 1, doi. 10.1371/journal.pone.0158295
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- Publication type:
- Article
Soluble Aβ oligomer-induced synaptopathy: c-Jun N-terminal kinase's role.
- Published in:
- Journal of Molecular Cell Biology, 2013, v. 5, n. 4, p. 277, doi. 10.1093/jmcb/mjt015
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- Publication type:
- Article
Crosstalk between JNK and SUMO Signaling Pathways: deSUMOylation Is Protective against H<sub>2</sub>O<sub>2</sub>-Induced Cell Injury.
- Published in:
- PLoS ONE, 2011, v. 6, n. 12, p. 1, doi. 10.1371/journal.pone.0028185
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- Publication type:
- Article
JNK Plays a Key Role in Tau Hyperphosphorylation in Alzheimer's Disease Models.
- Published in:
- Journal of Alzheimer's Disease, 2011, v. 26, n. 2, p. 315, doi. 10.3233/JAD-2011-110320
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- Publication type:
- Article
Abeta 1-40 and 1-42 oligomer-mediated memory impairment does not require cellular prion protein
- Published in:
- 2010
- By:
- Publication type:
- Abstract
JNK Contributes to Hif-1α Regulation in Hypoxic Neurons.
- Published in:
- Molecules, 2010, v. 15, n. 1, p. 114, doi. 10.3390/molecules15010114
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- Publication type:
- Article