Found: 16
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Diet-microbiome interactions promote enteric nervous system resilience following spinal cord injury.
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- NPJ Biofilms & Microbiomes, 2024, v. 10, n. 1, p. 1, doi. 10.1038/s41522-024-00556-y
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- Article
CRISPR-Cas systems: new players in gene regulation and bacterial physiology.
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- Frontiers in Cellular & Infection Microbiology, 2014, v. 4, n. 4, p. 1, doi. 10.3389/fcimb.2014.00037
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- Article
Repression of bacterial lipoprotein production by Francisella novicida facilitates evasion of innate immune recognition.
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- Cellular Microbiology, 2012, v. 14, n. 10, p. 1531, doi. 10.1111/j.1462-5822.2012.01816.x
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- Article
Cas9-dependent endogenous gene regulation is required for bacterial virulence.
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- Biochemical Society Transactions, 2013, v. 41, n. 6, p. 1407, doi. 10.1042/BST20130163
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- Article
Alternative Roles for CRISPR/Cas Systems in Bacterial Pathogenesis.
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- PLoS Pathogens, 2013, v. 9, n. 10, p. 1, doi. 10.1371/journal.ppat.1003621
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- Article
Defining Dysbiosis in Disorders of Movement and Motivation.
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- Journal of Neuroscience, 2018, v. 38, n. 44, p. 9414, doi. 10.1523/JNEUROSCI.1672-18.2018
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- Article
Soluble TNF mediates high‐fat and high‐carbohydrate diet–induced inflammation, alterations in peripheral blood and brain immunophenotype, and gut microbiome in a mouse model of amyloid pathology: Molecular and cell biology/neuroinflammation
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2020, v. 16, n. 11, p. 1, doi. 10.1002/alz.040436
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- Article
Corrigendum: A CRISPR/Cas system mediates bacterial innate immune evasion and virulence.
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- Nature, 2013, v. 501, n. 7466, p. 262, doi. 10.1038/nature12498
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- Article
A CRISPR/Cas system mediates bacterial innate immune evasion and virulence.
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- Nature, 2013, v. 497, n. 7448, p. 254, doi. 10.1038/nature12048
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- Article
Metagenomics of Parkinson's disease implicates the gut microbiome in multiple disease mechanisms.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-022-34667-x
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- Article
Soluble TNF mediates amyloid-independent, diet-induced alterations to immune and neuronal functions in an Alzheimer's disease mouse model.
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- Frontiers in Cellular Neuroscience, 2023, v. 17, p. 1, doi. 10.3389/fncel.2023.895017
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- Article
Circulating Extracellular Vesicles as a Potential Mediator of Synuclein Pathology in the Gut.
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- Movement Disorders, 2024, v. 39, n. 1, p. 3, doi. 10.1002/mds.29673
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- Article
Population fraction of Parkinson's disease attributable to preventable risk factors.
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- NPJ Parkinson's Disease, 2023, v. 9, n. 1, p. 1, doi. 10.1038/s41531-023-00603-z
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- Article
Exploiting CRISPR/ Cas systems for biotechnology.
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- BioEssays, 2014, v. 36, n. 1, p. 34, doi. 10.1002/bies.201300135
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- Article
Blocking soluble TNF decreases diet‐induced hyperglycemia and modulates immune alterations in 5xFAD animal model of Alzheimer's disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.067954
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- Article
The role of soluble TNF in mediating immune and metabolic alterations in a mouse model of amyloid‐beta pathology.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.055753
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- Article