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Intraneuronal amyloid-beta oligomers cause cell death via endoplasmic reticulum stress, endosomal/lysosomal leakage, and mitochondrial dysfunction in vivo
Published in:
2011
By:
- Umeda, Tomohiro;
- Tomiyama, Takami;
- Sakama, Naomi;
- Tanaka, Saya;
- Lambert, Mary;
- Klein, William;
- Mori, Hiroshi
Publication type:
Abstract
Intraneuronal accumulation of Aβ oligomers causes abnormal tau phosphorylation, glial activation and neuronal loss in transgenic mice with APP E693Δ mutation
Published in:
2010
By:
- Tomiyama, Takami;
- Umeda, Tomohiro;
- Sakama, Naomi;
- Lambert, Mary P.;
- Klein, William L.;
- Mori, Hiroshi
Publication type:
Abstract
Intraneuronal amyloid β oligomers cause cell death via endoplasmic reticulum stress, endosomal/lysosomal leakage, and mitochondrial dysfunction in vivo.
Published in:
Journal of Neuroscience Research, 2011, v. 89, n. 7, p. 1031, doi. 10.1002/jnr.22640
By:
- Umeda, Tomohiro;
- Tomiyama, Takami;
- Sakama, Naomi;
- Tanaka, Saya;
- Lambert, Mary P.;
- Klein, William L.;
- Mori, Hiroshi
Publication type:
Article
A Mouse Model of Amyloid β Oligomers: Their Contribution to Synaptic Alteration, Abnormal Tau Phosphorylation, Glial Activation, and Neuronal Loss In Vivo.
Published in:
Journal of Neuroscience, 2010, v. 30, n. 14, p. 4845, doi. 10.1523/JNEUROSCI.5825-09.2010
By:
- Tomiyama, Takami;
- Matsuyama, Shogo;
- Iso, Hiroyuki;
- Umeda, Tomohiro;
- Takuma, Hiroshi;
- Ohnishi, Kiyouhisa;
- Ishibashi, Kenichi;
- Teraoka, Rie;
- Sakama, Naomi;
- Yamashita, Takenari;
- Nishitsuji, Kazuchika;
- Ito, Kazuhiro;
- Shimada, Hiroyuki;
- Lambert, Mary P.;
- Klein, William L.;
- Mori1, Hiroshi
Publication type:
Article

Found: 4

Intraneuronal amyloid-beta oligomers cause cell death via endoplasmic reticulum stress, endosomal/lysosomal leakage, and mitochondrial dysfunction in vivo

Intraneuronal accumulation of Aβ oligomers causes abnormal tau phosphorylation, glial activation and neuronal loss in transgenic mice with APP E693Δ mutation

Intraneuronal amyloid β oligomers cause cell death via endoplasmic reticulum stress, endosomal/lysosomal leakage, and mitochondrial dysfunction in vivo.

A Mouse Model of Amyloid β Oligomers: Their Contribution to Synaptic Alteration, Abnormal Tau Phosphorylation, Glial Activation, and Neuronal Loss In Vivo.