Found: 17
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Artificial sweeteners inhibit multidrug‐resistant pathogen growth and potentiate antibiotic activity.
- Published in:
- EMBO Molecular Medicine, 2023, v. 15, n. 1, p. 1, doi. 10.15252/emmm.202216397
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- Article
A Fork Trap in the Chromosomal Termination Area Is Highly Conserved across All Escherichia coli Phylogenetic Groups.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 15, p. 7928, doi. 10.3390/ijms22157928
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- Article
Chromosomal over-replication in Escherichia coli recG cells is triggered by replication fork fusion and amplified if replichore symmetry is disturbed.
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- Nucleic Acids Research, 2018, v. 46, n. 15, p. 7701, doi. 10.1093/nar/gky566
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- Article
Shaping the landscape of the Escherichia coli chromosome: replication-transcription encounters in cells with an ectopic replication origin.
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- Nucleic Acids Research, 2015, v. 43, n. 16, p. 7865, doi. 10.1093/nar/gkv704
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- Article
Cellular location and activity of Escherichia coli RecG proteins shed light on the function of its structurally unresolved C-terminus.
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- Nucleic Acids Research, 2015, v. 43, n. 9, p. 5702, doi. 10.1093/nar/gku228
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- Article
Cellular location and activity of Escherichia coli RecG proteins shed light on the function of its structurally unresolved C-terminus.
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- Nucleic Acids Research, 2014, v. 42, n. 9, p. 5702, doi. 10.1093/nar/gku228
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- Article
Localization of an accessory helicase at the replisome is critical in sustaining efficient genome duplication.
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- Nucleic Acids Research, 2011, v. 39, n. 3, p. 949, doi. 10.1093/nar/gkq889
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- Article
Replication fork collisions cause pathological chromosomal amplification in cells lacking RecG DNA translocase.
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- Molecular Microbiology, 2009, v. 74, n. 4, p. 940, doi. 10.1111/j.1365-2958.2009.06909.x
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- Article
Pathological replication in cells lacking RecG DNA translocase.
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- Molecular Microbiology, 2009, v. 73, n. 3, p. 352, doi. 10.1111/j.1365-2958.2009.06773.x
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- Article
Interplay between chromosomal architecture and termination of DNA replication in bacteria.
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- Frontiers in Microbiology, 2023, p. 1, doi. 10.3389/fmicb.2023.1180848
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- Article
Avoiding chromosome pathology when replication forks collide.
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- Nature, 2013, v. 500, n. 7464, p. 608, doi. 10.1038/nature12312
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- Article
Cas1–Cas2 physically and functionally interacts with DnaK to modulate CRISPR Adaptation.
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- Nucleic Acids Research, 2023, v. 51, n. 13, p. 6914, doi. 10.1093/nar/gkad473
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- Article
Direct removal of RNA polymerase barriers to replication by accessory replicative helicases.
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- Nucleic Acids Research, 2019, v. 47, n. 10, p. 5100, doi. 10.1093/nar/gkz170
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- Article
role for 3′ exonucleases at the final stages of chromosome duplication in Escherichia coli.
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- Nucleic Acids Research, 2019, v. 47, n. 4, p. 1847, doi. 10.1093/nar/gky1253
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- Article
Origins Left, Right, and Centre: Increasing the Number of Initiation Sites in the Escherichia coli Chromosome.
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- Genes, 2018, v. 9, n. 8, p. 376, doi. 10.3390/genes9080376
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- Article
Replication Termination: Containing Fork Fusion-Mediated Pathologies in Escherichia coli.
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- Genes, 2016, v. 7, n. 8, p. 40, doi. 10.3390/genes7080040
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- Article
Too Much of a Good Thing: How Ectopic DNA Replication Affects Bacterial Replication Dynamics.
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- Frontiers in Microbiology, 2020, p. 1, doi. 10.3389/fmicb.2020.00534
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- Article