Works by Ruberti, Samantha
Results: 5
Calreticulin Ins5 and Del52 mutations impair unfolded protein and oxidative stress responses in K562 cells expressing CALR mutants.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-46843-z
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- Article
Role of TGF‐β1/miR‐382‐5p/SOD2 axis in the induction of oxidative stress in CD34+ cells from primary myelofibrosis.
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- Molecular Oncology, 2018, v. 12, n. 12, p. 2102, doi. 10.1002/1878-0261.12387
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- Article
Role of miR-34a-5p in Hematopoietic Progenitor Cells Proliferation and Fate Decision: Novel Insights into the Pathogenesis of Primary Myelofibrosis.
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- International Journal of Molecular Sciences, 2017, v. 18, n. 1, p. 145, doi. 10.3390/ijms18010145
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- Article
Integrative analysis of copy number and gene expression data suggests novel pathogenetic mechanisms in primary myelofibrosis.
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- International Journal of Cancer, 2016, v. 138, n. 7, p. 1657, doi. 10.1002/ijc.29920
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- Article
Abnormal expression patterns of WT1-as, MEG3 and ANRIL long non-coding RNAs in CD34+ cells from patients with primary myelofibrosis and their clinical correlations.
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- Leukemia & Lymphoma, 2015, v. 56, n. 2, p. 492, doi. 10.3109/10428194.2014.910661
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- Article