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Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide-Associated Ulcerative Colitis through JNK Inactivation and Chromatin Binding.
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- Oxidative Medicine & Cellular Longevity, 2017, p. 1, doi. 10.1155/2017/9303158
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- Article
Repeated H<sub>2</sub>O<sub>2</sub> exposure drives cell cycle progression in an in vitro model of ulcerative colitis.
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- Journal of Cellular & Molecular Medicine, 2013, v. 17, n. 12, p. 1619, doi. 10.1111/jcmm.12150
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- Article
ATF2 knockdown reinforces oxidative stress-induced apoptosis in TE7 cancer cells.
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- Journal of Cellular & Molecular Medicine, 2013, v. 17, n. 8, p. 976, doi. 10.1111/jcmm.12071
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- Article
Non-apoptotic function of caspases in a cellular model of hydrogen peroxide-associated colitis.
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- Journal of Cellular & Molecular Medicine, 2013, v. 17, n. 7, p. 901, doi. 10.1111/jcmm.12079
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- Article
Cutting edge: Chk1 directs senescence and mitotic catastrophe in recovery from G<sub>2</sub> checkpoint arrest.
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- Journal of Cellular & Molecular Medicine, 2011, v. 15, n. 7, p. 1528, doi. 10.1111/j.1582-4934.2010.01143.x
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- Article
Diagnostic, Therapy and Complications in Acute Appendicitis of 19,749 Cases Based on Routine Data: A Retrospective Multicenter Observational Study.
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- Journal of Clinical Medicine, 2022, v. 11, n. 15, p. 4495, doi. 10.3390/jcm11154495
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- Article