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Amyloid-β Peptide Fibrils Induce Nitro-Oxidative Stress in Neuronal Cells.
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- Journal of Alzheimer's Disease, 2010, v. 22, n. 2, p. 641, doi. 10.3233/JAD-2010-100474
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- Article
Activation of PKR Causes Amyloid ß-Peptide Accumulation via De-Repression of BACE1 Expression.
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- PLoS ONE, 2011, v. 6, n. 6, p. 1, doi. 10.1371/journal.pone.0021456
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- Article
Paradoxical neuronal hyperexcitability in a mouse model of mitochondrial pyruvate import deficiency.
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- eLife, 2022, p. 1, doi. 10.7554/eLife.72595
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- Article
Astrocytic release of ATP through type 2 inositol 1,4,5‐trisphosphate receptor calcium signaling and social dominance behavior in mice.
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- European Journal of Neuroscience, 2021, v. 53, n. 9, p. 2973, doi. 10.1111/ejn.14892
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- Article
Glutathione in the nucleus accumbens regulates motivation to exert reward-incentivized effort.
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- eLife, 2022, p. 1, doi. 10.7554/eLife.77791
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- Article
Wnt5a promotes hippocampal postsynaptic development and GluN2B-induced expression via the eIF2α HRI kinase.
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- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-86708-y
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- Article
Wnt Signaling Prevents the Aβ Oligomer-Induced Mitochondrial Permeability Transition Pore Opening Preserving Mitochondrial Structure in Hippocampal Neurons.
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- PLoS ONE, 2017, v. 12, n. 1, p. 1, doi. 10.1371/journal.pone.0168840
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- Article
Nitro-Oxidative Stress after Neuronal Ischemia Induces Protein Nitrotyrosination and Cell Death.
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- Oxidative Medicine & Cellular Longevity, 2013, v. 2013, p. 1, doi. 10.1155/2013/826143
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- Article
Wnt-7a Stimulates Dendritic Spine Morphogenesis and PSD-95 Expression Through Canonical Signaling.
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- Molecular Neurobiology, 2019, v. 56, n. 3, p. 1870, doi. 10.1007/s12035-018-1162-1
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- Article
Modulation of the NMDA Receptor Through Secreted Soluble Factors.
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- Molecular Neurobiology, 2016, v. 53, n. 1, p. 299, doi. 10.1007/s12035-014-9009-x
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- Article
The Gαo Activator Mastoparan-7 Promotes Dendritic Spine Formation in Hippocampal Neurons.
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- Neural Plasticity, 2015, v. 2015, p. 1, doi. 10.1155/2016/4258171
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- Article