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Inflammasome-Mediated Neuronal-Microglial Crosstalk: a Therapeutic Substrate for the Familial C9orf72 Variant of Frontotemporal Dementia/Amyotrophic Lateral Sclerosis.
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- Molecular Neurobiology, 2023, v. 60, n. 7, p. 4004, doi. 10.1007/s12035-023-03315-w
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COVID‐19 and Alzheimer's disease: Meninges‐mediated neuropathology.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.056418
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Causal effects of microglia‐mediated innate immune responses in the pathogenesis of c9orf72 frontotemporal dementia and amyotrophic lateral sclerosis.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.051865
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- Article
Neuronal Pentraxin 1 Promotes Hypoxic-Ischemic Neuronal Injury by Impairing Mitochondrial Biogenesis via Interactions With Active Bax[6A7] and Mitochondrial Hexokinase II.
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- ASN Neuro (Sage Publications, Ltd.), 2021, p. 1, doi. 10.1177/17590914211012888
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- Article
Neuronal Pentraxin 1 Promotes Hypoxic-Ischemic Neuronal Injury by Impairing Mitochondrial Biogenesis via Interactions With Active Bax[6A7] and Mitochondrial Hexokinase II.
- Published in:
- ASN Neuro (Sage Publications, Ltd.), 2021, p. 1, doi. 10.1177/17590914211012888
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- Publication type:
- Article
Presenilin 1 is necessary for neuronal, but not glial, EGFR expression and neuroprotection via γ-secretase-independent transcriptional mechanisms.
- Published in:
- FASEB Journal, 2015, v. 29, n. 9, p. 3702, doi. 10.1096/fj.15-270645
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- Article