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Loss of Tmem106b is unable to ameliorate frontotemporal dementia-like phenotypes in an AAV mouse model of <italic>C9ORF72</italic>-repeat induced toxicity.
Published in:
Acta Neuropathologica Communications, 2018, v. 6, n. 1, p. N.PAG, doi. 10.1186/s40478-018-0545-x
By:
- Nicholson, Alexandra M.;
- Zhou, Xiaolai;
- Perkerson, Ralph B.;
- Parsons, Tammee M.;
- Chew, Jeannie;
- Brooks, Mieu;
- DeJesus-Hernandez, Mariely;
- Finch, NiCole A.;
- Matchett, Billie J.;
- Kurti, Aishe;
- Jansen-West, Karen R.;
- Perkerson, Emilie;
- Daughrity, Lillian;
- Castanedes-Casey, Monica;
- Rousseau, Linda;
- Phillips, Virginia;
- Hu, Fenghua;
- Gendron, Tania F.;
- Murray, Melissa E.;
- Dickson, Dennis W.
Publication type:
Article
Poly(GP) proteins are a useful pharmacodynamic marker for C9ORF72-associated amyotrophic lateral sclerosis.
Published in:
Science Translational Medicine, 2017, v. 9, n. 383, p. 1, doi. 10.1126/scitranslmed.aai7866
By:
- Gendron, Tania F.;
- Chew, Jeannie;
- Stankowski, Jeannette N.;
- Hayes, Lindsey R.;
- Yong-Jie Zhang;
- Prudencio, Mercedes;
- Carlomagno, Yari;
- Daughrity, Lillian M.;
- Jansen-West, Karen;
- Perkerson, Emilie A.;
- O'Raw, Aliesha;
- Cook, Casey;
- Pregent, Luc;
- Belzil, Veronique;
- van Blitterswijk, Marka;
- Tabassian, Lilia J.;
- Lee, Chris W.;
- Mei Yue;
- Jimei Tong;
- Yuping Song
Publication type:
Article
Tau deposition drives neuropathological, inflammatory and behavioral abnormalities independently of neuronal loss in a novel mouse model.
Published in:
Human Molecular Genetics, 2015, v. 24, n. 21, p. 6198, doi. 10.1093/hmg/ddv336
By:
- Cook, Casey;
- Kang, Silvia S.;
- Carlomagno, Yari;
- Wen-Lang Lin;
- Mei Yue;
- Aishe Kurti;
- Mitsuru Shinohara;
- Jansen-West, Karen;
- Perkerson, Emilie;
- Castanedes-Casey, Monica;
- Rousseau, Linda;
- Phillips, Virginia;
- Guojun Bu;
- Dickson, Dennis W.;
- Petrucelli, Leonard;
- Fryer, John D.
Publication type:
Article

Found: 3

Loss of Tmem106b is unable to ameliorate frontotemporal dementia-like phenotypes in an AAV mouse model of <italic>C9ORF72</italic>-repeat induced toxicity.

Poly(GP) proteins are a useful pharmacodynamic marker for C9ORF72-associated amyotrophic lateral sclerosis.

Tau deposition drives neuropathological, inflammatory and behavioral abnormalities independently of neuronal loss in a novel mouse model.