Works by Pérez‐Navarro, Esther
Results: 36
Caffeine-mediated BDNF release regulates long-term synaptic plasticity through activation of IRS2 signaling.
- Published in:
- 2017
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- Publication type:
- journal article
RTP801/REDD1 Is Involved in Neuroinflammation and Modulates Cognitive Dysfunction in Huntington's Disease.
- Published in:
- Biomolecules (2218-273X), 2022, v. 12, n. 1, p. 34, doi. 10.3390/biom12010034
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- Publication type:
- Article
Synaptic RTP801 contributes to motor-learning dysfunction in Huntington’s disease.
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- Cell Death & Disease, 2020, v. 11, n. 7, p. 1, doi. 10.1038/s41419-020-02775-5
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- Publication type:
- Article
Early Down-Regulation of PKCδ as a Pro-Survival Mechanism in Huntington's Disease.
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- NeuroMolecular Medicine, 2014, v. 16, n. 1, p. 25, doi. 10.1007/s12017-013-8248-8
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- Publication type:
- Article
Increased 5-Methylcytosine and Decreased 5-Hydroxymethylcytosine Levels are Associated with Reduced Striatal AR Levels in Huntington's Disease.
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- NeuroMolecular Medicine, 2013, v. 15, n. 2, p. 295, doi. 10.1007/s12017-013-8219-0
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- Publication type:
- Article
Brain-derived neurotrophic factor prevents changes in Bcl-2 family members and caspase-3 activation induced by excitotoxicity in the striatum.
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- Journal of Neurochemistry, 2005, v. 92, n. 3, p. 678, doi. 10.1111/j.1471-4159.2004.02904.x
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- Publication type:
- Article
Brain-Derived Neurotrophic Factor, Neurotrophin-3, and Neurotrophin-4/5 Prevent the Death of Striatal Projection Neurons in a Rodent Model of Huntington's Disease.
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- Journal of Neurochemistry, 2000, v. 75, n. 5, p. 2190, doi. 10.1046/j.1471-4159.2000.0752190.x
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- Publication type:
- Article
Involvement of Nerve Growth Factor and Its Receptor in the Regulation of the Cholinergic Function in Aged Rats.
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- Journal of Neurochemistry, 1991, v. 57, n. 5, p. 1483, doi. 10.1111/j.1471-4159.1991.tb06342.x
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- Publication type:
- Article
Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease.
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- Human Molecular Genetics, 2011, v. 20, n. 21, p. 4232, doi. 10.1093/hmg/ddr351
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- Publication type:
- Article
Preserved VPS13A distribution and expression in Huntington's disease: divergent mechanisms of action for similar movement disorders?
- Published in:
- Frontiers in Neuroscience, 2024, p. 1, doi. 10.3389/fnins.2024.1394478
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- Publication type:
- Article
Cognitive dysfunction in Huntington's disease: mechanisms and therapeutic strategies beyond BDNF.
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- Brain Pathology, 2016, v. 26, n. 6, p. 752, doi. 10.1111/bpa.12432
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- Publication type:
- Article
Motor skill learning modulates striatal extracellular vesicles' content in a mouse model of Huntington's disease.
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- Cell Communication & Signaling, 2024, v. 22, n. 1, p. 1, doi. 10.1186/s12964-024-01693-9
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- Publication type:
- Article
Increased Phospho‐AKT in Blood Cells from LRRK2 G2019S Mutation Carriers.
- Published in:
- Annals of Neurology, 2022, v. 92, n. 5, p. 888, doi. 10.1002/ana.26469
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- Publication type:
- Article
Down-regulation of BDNF in cell and animal models increases striatal-enriched protein tyrosine phosphatase 61 ( STEP<sub>61</sub>) levels.
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- Journal of Neurochemistry, 2016, v. 136, n. 2, p. 285, doi. 10.1111/jnc.13295
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- Publication type:
- Article
Calcineurin is involved in the early activation of NMDA-mediated cell death in mutant huntingtin knock-in striatal cells.
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- Journal of Neurochemistry, 2008, v. 105, n. 5, p. 1596, doi. 10.1111/j.1471-4159.2008.05252.x
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- Publication type:
- Article
PDE10 inhibition increases GluA1 and CREB phosphorylation and improves spatial and recognition memories in a Huntington's disease mouse model.
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- Hippocampus, 2013, v. 23, n. 8, p. 684, doi. 10.1002/hipo.22128
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- Publication type:
- Article
Lamin B1 and nuclear morphology in peripheral cells as new potential biomarkers to follow treatment response in Huntington's disease.
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- Clinical & Translational Medicine, 2023, v. 13, n. 2, p. 1, doi. 10.1002/ctm2.1154
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- Publication type:
- Article
Increased translation as a novel pathogenic mechanism in Huntington's disease.
- Published in:
- 2019
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- Publication type:
- journal article
Proteolytic Degradation of Hippocampal STEP<sub>61</sub> in LTP and Learning.
- Published in:
- Molecular Neurobiology, 2019, v. 56, n. 2, p. 1475, doi. 10.1007/s12035-018-1170-1
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- Publication type:
- Article
Increased Levels of Rictor Prevent Mutant Huntingtin-Induced Neuronal Degeneration.
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- Molecular Neurobiology, 2018, v. 55, n. 10, p. 7728, doi. 10.1007/s12035-018-0956-5
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- Publication type:
- Article
BDNF Induces Striatal-Enriched Protein Tyrosine Phosphatase 61 Degradation Through the Proteasome.
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- Molecular Neurobiology, 2016, v. 53, n. 6, p. 4261, doi. 10.1007/s12035-015-9335-7
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- Publication type:
- Article
RTP801 Is Involved in Mutant Huntingtin-Induced Cell Death.
- Published in:
- Molecular Neurobiology, 2016, v. 53, n. 5, p. 2857, doi. 10.1007/s12035-015-9166-6
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- Publication type:
- Article
Sustained Increase of PKA Activity in the Postcommissural Putamen of Dyskinetic Monkeys.
- Published in:
- Molecular Neurobiology, 2014, v. 50, n. 3, p. 1131, doi. 10.1007/s12035-014-8688-7
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- Publication type:
- Article
Huntington's disease brain-derived small RNAs recapitulate associated neuropathology in mice.
- Published in:
- Acta Neuropathologica, 2021, v. 141, n. 4, p. 565, doi. 10.1007/s00401-021-02272-9
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- Publication type:
- Article
Glial cell line‐derived neurotrophic factor promotes the arborization of cultured striatal neurons through the p42/p44 mitogen‐activated protein kinase pathway.
- Published in:
- Journal of Neuroscience Research, 2006, v. 83, n. 1, p. 68
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- Publication type:
- Article
Targeting CAG repeat RNAs reduces Huntington's disease phenotype independently of huntingtin levels.
- Published in:
- 2016
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- Publication type:
- journal article
Neuron type‐specific increase in lamin B1 contributes to nuclear dysfunction in Huntington's disease.
- Published in:
- EMBO Molecular Medicine, 2021, v. 13, n. 2, p. 1, doi. 10.15252/emmm.202012105
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- Publication type:
- Article
RTP801 mediates transneuronal toxicity in culture via extracellular vesicles.
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- Journal of Extracellular Vesicles, 2023, v. 12, n. 11, p. 1, doi. 10.1002/jev2.12378
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- Publication type:
- Article
Neuron‐derived extracellular vesicles contain synaptic proteins, promote spine formation, activate TrkB‐mediated signalling and preserve neuronal complexity.
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- Journal of Extracellular Vesicles, 2023, v. 12, n. 9, p. 1, doi. 10.1002/jev2.12355
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- Publication type:
- Article
Decreased glycogen synthase kinase-3 levels and activity contribute to Huntington's disease.
- Published in:
- Human Molecular Genetics, 2015, v. 24, n. 17, p. 5040, doi. 10.1093/hmg/ddv224
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- Publication type:
- Article
RTP801 interacts with the tRNA ligase complex and dysregulates its RNA ligase activity in Alzheimer's disease.
- Published in:
- Nucleic Acids Research, 2024, v. 52, n. 18, p. 11158, doi. 10.1093/nar/gkae776
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- Publication type:
- Article
Nerve Growth Factor and Basic Fibroblast Growth Factor Protect Cholinergic Neurons Against Quinolinic Acid Excitotoxicity in Rat Neostriatum.
- Published in:
- European Journal of Neuroscience, 1994, v. 6, n. 5, p. 706, doi. 10.1111/j.1460-9568.1994.tb00982.x
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- Publication type:
- Article
Striatal-Enriched Protein Tyrosine Phosphatase Expression and Activity in Huntington’s Disease: A STEP in the Resistance to Excitotoxicity.
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- Journal of Neuroscience, 2011, v. 31, n. 22, p. 8150, doi. 10.1523/JNEUROSCI.3446-10.2011
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- Publication type:
- Article
Bax and Calpain Mediate Excitotoxic Oligodendrocyte Death Induced by Activation of Both AMPA and Kainate Receptors.
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- Journal of Neuroscience, 2011, v. 31, n. 8, p. 2996, doi. 10.1523/JNEUROSCI.5578-10.2011
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- Publication type:
- Article
THE NOVEL AMPA RECEPTOR POSITIVE ALLOSTERIC MODULATOR S 47445 RESCUES IN VIVO CA3-CA1 LONG-TERM POTENTIATION AND STRUCTURAL SYNAPTIC CHANGES IN MIDDLE-AGED MICE.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2017, v. 13, p. P1270, doi. 10.1016/j.jalz.2017.06.1900
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- Publication type:
- Article
Regulation of Hippocampal cGMP Levels as a Candidate to Treat Cognitive Deficits in Huntington’s Disease.
- Published in:
- PLoS ONE, 2013, v. 8, n. 9, p. 1, doi. 10.1371/journal.pone.0073664
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- Publication type:
- Article