CCL5 Induces a Sarcopenic-like Phenotype via the CCR5 Receptor.
- Published in:
- Antioxidants, 2025, v. 14, n. 1, p. 84, doi. 10.3390/antiox14010084
- By:
- Publication type:
- Article
Works by Orozco-Aguilar, Josué
Results: 11
1
2
Author Correction: Entry of spores into intestinal epithelial cells contributes to recurrence of Clostridioides difficile infection.
- Published in:
- 2022
- By:
- Publication type:
- Correction Notice
3
Role of Oxidative Stress in Hepatic and Extrahepatic Dysfunctions during Nonalcoholic Fatty Liver Disease (NAFLD).
- Published in:
- Oxidative Medicine & Cellular Longevity, 2020, p. 1, doi. 10.1155/2020/1617805
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- Publication type:
- Article
4
SARS-CoV-2/Renin–Angiotensin System: Deciphering the Clues for a Couple with Potentially Harmful Effects on Skeletal Muscle.
- Published in:
- International Journal of Molecular Sciences, 2020, v. 21, n. 21, p. 7904, doi. 10.3390/ijms21217904
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- Publication type:
- Article
5
Cholic and deoxycholic acids induce mitochondrial dysfunction, impaired biogenesis and autophagic flux in skeletal muscle cells.
- Published in:
- Biological Research, 2023, v. 56, n. 1, p. 1, doi. 10.1186/s40659-023-00436-3
- By:
- Publication type:
- Article
6
Ursodeoxycholic acid induces sarcopenia associated with decreased protein synthesis and autophagic flux.
- Published in:
- Biological Research, 2023, v. 56, n. 1, p. 1, doi. 10.1186/s40659-023-00431-8
- By:
- Publication type:
- Article
7
Redox-Dependent Effects in the Physiopathological Role of Bile Acids.
- Published in:
- Oxidative Medicine & Cellular Longevity, 2021, p. 1, doi. 10.1155/2021/4847941
- By:
- Publication type:
- Article
8
In vivo animal models confirm an increased virulence potential and pathogenicity of the NAP1/RT027/ST01 genotype within the Clostridium difficile MLST Clade 2.
- Published in:
- Gut Pathogens, 2020, v. 12, n. 1, p. N.PAG, doi. 10.1186/s13099-020-00383-4
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- Publication type:
- Article
9
Novel Clade C-I Clostridium difficile strains escape diagnostic tests, differ in pathogenicity potential and carry toxins on extrachromosomal elements.
- Published in:
- Scientific Reports, 2018, v. 8, n. 1, p. 1, doi. 10.1038/s41598-018-32390-6
- By:
- Publication type:
- Article
10
Entry of spores into intestinal epithelial cells contributes to recurrence of Clostridioides difficile infection.
- Published in:
- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-21355-5
- By:
- Publication type:
- Article
11
Toxin B Variants from Clostridium difficile Strains VPI 10463 and NAP1/027 Share Similar Substrate Profile and Cellular Intoxication Kinetics but Use Different Host Cell Entry Factors.
- Published in:
- Toxins, 2019, v. 11, n. 6, p. 348, doi. 10.3390/toxins11060348
- By:
- Publication type:
- Article