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Correction: Type I Interferon Receptor Deficiency in Dendritic Cells Facilitates Systemic Murine Norovirus Persistence Despite Enhanced Adaptive Immunity.
- Published in:
- 2017
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- Publication type:
- Correction Notice
Type I Interferon Receptor Deficiency in Dendritic Cells Facilitates Systemic Murine Norovirus Persistence Despite Enhanced Adaptive Immunity.
- Published in:
- PLoS Pathogens, 2016, v. 12, n. 6, p. 1, doi. 10.1371/journal.ppat.1005684
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- Publication type:
- Article
Murine norovirus protein NS1/2 aspartate to glutamate mutation, sufficient for persistence, reorients side chain of surface exposed tryptophan within a novel structured domain.
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- Proteins, 2014, v. 82, n. 7, p. 1200, doi. 10.1002/prot.24484
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- Article
Homeostatic interferon-lambda response to bacterial microbiota stimulates preemptive antiviral defense within discrete pockets of intestinal epithelium.
- Published in:
- eLife, 2022, p. 1, doi. 10.7554/eLife.74072
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- Publication type:
- Article
Norovirus evolution in immunodeficient mice reveals potentiated pathogenicity via a single nucleotide change in the viral capsid.
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- PLoS Pathogens, 2021, v. 17, n. 3, p. 1, doi. 10.1371/journal.ppat.1009402
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- Article
Caspase-mediated cleavage of murine norovirus NS1/2 potentiates apoptosis and is required for persistent infection of intestinal epithelial cells.
- Published in:
- PLoS Pathogens, 2019, v. 15, n. 7, p. 1, doi. 10.1371/journal.ppat.1007940
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- Publication type:
- Article
Interferon Lambda in the Pathogenesis of Inflammatory Bowel Diseases.
- Published in:
- Frontiers in Immunology, 2021, v. 12, p. 1, doi. 10.3389/fimmu.2021.767505
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- Article