Found: 14
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Inhibition of the ISR abrogates mGluR5-dependent long-term depression and spatial memory deficits in a rat model of Alzheimer's disease.
- Published in:
- Translational Psychiatry, 2022, v. 12, n. 1, p. 1, doi. 10.1038/s41398-022-01862-9
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- Article
mGlu5 receptors and cellular prion protein mediate amyloid-β-facilitated synaptic long-term depression in vivo.
- Published in:
- Nature Communications, 2014, v. 5, n. 3, p. 3374, doi. 10.1038/ncomms4374
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- Article
Alzheimer's disease Aβ assemblies mediating rapid disruption of synaptic plasticity and memory.
- Published in:
- Molecular Brain, 2012, v. 5, n. 1, p. 25, doi. 10.1186/1756-6606-5-25
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- Article
S 44858, a positive allosteric modulator of ampa receptors and GluN2B selective nmda receptors antagonist, prevents the disruption of synaptic plasticity induced by Aβ oligomers.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P845, doi. 10.1016/j.jalz.2015.06.853
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- Article
Alzheimer's disease human brain beta-amyloid-containing extracts inhibit hippocampal long-term potentiation in rats in vivo: Relationship between soluble and insoluble preparations
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- 2012
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- Abstract
Switching off LTP: mGlu and NMDA Receptor–Dependent Novelty Exploration–Induced Depotentiation in the Rat Hippocampus.
- Published in:
- Cerebral Cortex, 2013, v. 23, n. 4, p. 932
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- Article
Death-associated protein kinase 1 is associated with cognitive dysfunction in major depressive disorder.
- Published in:
- Neural Regeneration Research, 2023, v. 18, n. 8, p. 1795, doi. 10.4103/1673-5374.361532
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- Publication type:
- Article
Do tau-synaptic long-term depression interactions in the hippocampus play a pivotal role in the progression of Alzheimer's disease?
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- Neural Regeneration Research, 2023, v. 18, n. 6, p. 1213, doi. 10.4103/1673-5374.360166
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- Publication type:
- Article
Alzheimer’s Disease Amyloid β-Protein and Synaptic Function.
- Published in:
- NeuroMolecular Medicine, 2010, v. 12, n. 1, p. 13, doi. 10.1007/s12017-009-8091-0
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- Article
Tau and Amyloid β Protein in Patient-Derived Aqueous Brain Extracts Act Concomitantly to Disrupt Long-Term Potentiation in Vivo.
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- Journal of Neuroscience, 2023, v. 43, n. 32, p. 5870, doi. 10.1523/JNEUROSCI.0082-23.2023
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- Article
Gamma‐patterned sensory stimulation reverses synaptic plasticity deficits in rat models of early Alzheimer's disease.
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- European Journal of Neuroscience, 2023, v. 58, n. 6, p. 3402, doi. 10.1111/ejn.16129
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- Publication type:
- Article
Pre-plaque Aß-Mediated Impairment of Synaptic Depotentiation in a Transgenic Rat Model of Alzheimer's Disease Amyloidosis.
- Published in:
- Frontiers in Neuroscience, 2019, p. 1, doi. 10.3389/fnins.2019.00861
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- Publication type:
- Article
Soluble amyloid-{beta} peptides potently disrupt hippocampal synaptic plasticity in the absence of cerebrovascular dysfunction in vivo.
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- Brain: A Journal of Neurology, 2008, v. 131, n. 9, p. 2414
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- Article
LTD‐inducing low frequency stimulation enhances p‐Tau181 and p‐Tau217 in an age‐dependent manner in live rats.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.064619
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- Publication type:
- Article