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A Longitudinal, Integrated, Clinical, Histological and mRNA Profiling Study of Resistance Exercise in Myositis.
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- Molecular Medicine, 2010, v. 16, n. 11/12, p. 455
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- Article
Characterization of the dystrophin‐associated protein complex by mass spectrometry.
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- Mass Spectrometry Reviews, 2024, v. 43, n. 1, p. 90, doi. 10.1002/mas.21823
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- Article
Annexin A1 Deficiency does not Affect Myofiber Repair but Delays Regeneration of Injured Muscles.
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- Scientific Reports, 2015, p. 18246, doi. 10.1038/srep18246
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Muscle myeloid type I interferon gene expression may predict therapeutic responses to rituximab in myositis patients.
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- Rheumatology, 2016, v. 55, n. 9, p. 1673, doi. 10.1093/rheumatology/kew213
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- Article
Ryanodine channel complex stabilizer compound S48168/ARM210 as a disease modifier in dystrophin-deficient mdx mice: proof-of-concept study and independent validation of efficacy.
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- FASEB Journal, 2018, v. 32, n. 2, p. 1025, doi. 10.1096/fj.201700182RRR
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- Article
Mitotic Asynchrony Induces Transforming Growth Factor-β1 Secretion from Airway Epithelium.
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- American Journal of Respiratory Cell & Molecular Biology, 2014, v. 51, n. 3, p. 363, doi. 10.1165/rcmb.2013-0396OC
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- Article
Death and Disability Reported with Cases of Vaccine Anaphylaxis Stratified by Administration Setting: An Analysis of the Vaccine Adverse Event Reporting System from 2017 to 2022.
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- Vaccines, 2023, v. 11, n. 2, p. 276, doi. 10.3390/vaccines11020276
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Urine proteomics by mass spectrometry identifies proteins involved in key pathogenic pathways in patients with juvenile dermatomyositis.
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- Rheumatology, 2023, v. 62, n. 9, p. 3161, doi. 10.1093/rheumatology/kead033
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A novel estrogen receptor 1: sphingomyelin phosphodiesterase acid-like 3B pathway mediates rituximab response in myositis patients.
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- Rheumatology, 2023, v. 62, n. 8, p. 2864, doi. 10.1093/rheumatology/keac687
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- Article
Discovery of serum protein biomarkers in the mdx mouse model and cross-species comparison to Duchenne muscular dystrophy patients.
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- Human Molecular Genetics, 2014, v. 23, n. 24, p. 6458, doi. 10.1093/hmg/ddu366
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- Article
Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophy.
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- Human Molecular Genetics, 2014, v. 23, n. 12, p. 3239, doi. 10.1093/hmg/ddu033
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- Article
Role of toll-like receptors in the pathogenesis of dystrophin-deficient skeletal and heart muscle.
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- Human Molecular Genetics, 2014, v. 23, n. 10, p. 2604, doi. 10.1093/hmg/ddt656
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- Article
Myoblasts and macrophages are required for therapeutic morpholino antisense oligonucleotide delivery to dystrophic muscle.
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- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-00924-7
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- Article
A Promising Future for Stem-Cell-Based Therapies in Muscular Dystrophies—In Vitro and In Vivo Treatments to Boost Cellular Engraftment.
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- International Journal of Molecular Sciences, 2019, v. 20, n. 21, p. 5433, doi. 10.3390/ijms20215433
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- Article
Loss of calpain3b in Zebrafish, a Model of Limb-Girdle Muscular Dystrophy, Increases Susceptibility to Muscle Defects Due to Elevated Muscle Activity.
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- Genes, 2023, v. 14, n. 2, p. 492, doi. 10.3390/genes14020492
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Sexual dimorphism in immune response genes as a function of puberty.
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- BMC Immunology, 2006, v. 7, p. 2, doi. 10.1186/1471-2172-7-2
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One-year Treatment of Morpholino Antisense Oligomer Improves Skeletal and Cardiac Muscle Functions in Dystrophic mdx Mice.
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- Molecular Therapy, 2011, v. 19, n. 3, p. 576, doi. 10.1038/mt.2010.288
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- Article
Replacing acid α-glucosidase in Pompe disease: recombinant and transgenic enzymes are equipotent, but neither completely clears glycogen from type II muscle fibers
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- Molecular Therapy, 2005, v. 11, n. 1, p. 48, doi. 10.1016/j.ymthe.2004.09.017
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Glycogen Stored in Skeletal but Not in Cardiac Muscle in Acid α-Glucosidase Mutant (Pompe) Mice Is Highly Resistant to Transgene-Encoded Human Enzyme
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- Molecular Therapy, 2002, v. 6, n. 5, p. 601, doi. 10.1006/mthe.2002.0716
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Muscle Weakness in Myositis: MicroRNA‐Mediated Dystrophin Reduction in a Myositis Mouse Model and Human Muscle Biopsies.
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- Arthritis & Rheumatology, 2020, v. 72, n. 7, p. 1170, doi. 10.1002/art.41215
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Endurance Exercise Improves Molecular Pathways of Aerobic Metabolism in Patients With Myositis.
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- Arthritis & Rheumatology, 2016, v. 68, n. 7, p. 1738, doi. 10.1002/art.39624
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- Article
Fibroadipogenic progenitors are responsible for muscle loss in limb girdle muscular dystrophy 2B.
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- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-10438-z
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- Article
Mitochondrial dysfunction and consequences in calpain-3-deficient muscle.
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- Skeletal Muscle, 2020, v. 10, n. 1, p. 1, doi. 10.1186/s13395-020-00254-1
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Sources of variability and effect of experimental approach on expression profiling data interpretation.
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- BMC Bioinformatics, 2002, v. 3, p. 4, doi. 10.1186/1471-2105-3-4
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Non-Invasive MRI and Spectroscopy of mdx Mice Reveal Temporal Changes in Dystrophic Muscle Imaging and in Energy Deficits.
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- PLoS ONE, 2014, v. 9, n. 11, p. 1, doi. 10.1371/journal.pone.0112477
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The Proton Pump Inhibitor Lansoprazole Improves the Skeletal Phenotype in Dystrophin Deficient <i>mdx</i> Mice.
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- PLoS ONE, 2013, v. 8, n. 7, p. 1, doi. 10.1371/journal.pone.0066617
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Daily Supplementation of D-ribose Shows No Therapeutic Benefits in the MHC-I Transgenic Mouse Model of Inflammatory Myositis.
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- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0065970
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- Article
Omigapil Treatment Decreases Fibrosis and Improves Respiratory Rate in dy<sup>2J</sup> Mouse Model of Congenital Muscular Dystrophy.
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- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0065468
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VBP15, a Glucocorticoid Analogue, Is Effective at Reducing Allergic Lung Inflammation in Mice
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- PLoS ONE, 2013, v. 8, n. 5, p. 1, doi. 10.1371/journal.pone.0063871
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Platelets Induce Apoptosis during Sepsis in a Contact-Dependent Manner That Is Inhibited by GPIIb/IIIa Blockade.
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- PLoS ONE, 2012, v. 7, n. 7, p. 1, doi. 10.1371/journal.pone.0041549
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Glucocorticoid-Treated Mice Are an Inappropriate Positive Control for Long-Term Preclinical Studies in the mdx Mouse.
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- PLoS ONE, 2012, v. 7, n. 4, p. 1, doi. 10.1371/journal.pone.0034204
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Characterization of Dysferlin Deficient SJL/J Mice to Assess Preclinical Drug Efficacy: Fasudil Exacerbates Muscle Disease Phenotype.
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- PLoS ONE, 2010, v. 5, n. 9, p. 1, doi. 10.1371/journal.pone.0012981
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Functional and Molecular Effects of Arginine Butyrate and Prednisone on Muscle and Heart in the mdx Mouse Model of Duchenne Muscular Dystrophy.
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- PLoS ONE, 2010, v. 5, n. 6, p. 1, doi. 10.1371/journal.pone.0011220
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Evaluation of Skeletal and Cardiac Muscle Function after Chronic Administration of Thymosin β-4 in the Dystrophin Deficient Mouse.
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- PLoS ONE, 2010, v. 5, n. 1, p. 1, doi. 10.1371/journal.pone.0008976
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Mitochondrial dysfunction and role of harakiri in the pathogenesis of myositis.
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- Journal of Pathology, 2019, v. 249, n. 2, p. 215, doi. 10.1002/path.5309
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Selective modulation through the glucocorticoid receptor ameliorates muscle pathology in mdx mice.
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- Journal of Pathology, 2013, v. 231, n. 2, p. 223, doi. 10.1002/path.4231
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- Article
The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: role of endogenous TLR ligands.
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- Journal of Pathology, 2013, v. 231, n. 2, p. 199, doi. 10.1002/path.4207
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- Article
NKG2A inhibits TH2 cell effector function in vitro.
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- BMC Pulmonary Medicine, 2007, v. 7, p. 1, doi. 10.1186/1471-2466-7-14
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Serum pharmacodynamic biomarkers for chronic corticosteroid treatment of children.
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- Scientific Reports, 2016, p. 31727, doi. 10.1038/srep31727
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Mechanism of action and therapeutic route for a muscular dystrophy caused by a genetic defect in lipid metabolism.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-022-29270-z
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- Article
Skeletal, cardiac, and respiratory muscle function and histopathology in the <italic>P448Lneo−</italic> mouse model of FKRP-deficient muscular dystrophy.
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- Skeletal Muscle, 2018, v. 8, n. 1, p. 1, doi. 10.1186/s13395-018-0158-x
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- Article
Elusive sources of variability of dystrophin rescue by exon skipping.
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- Skeletal Muscle, 2015, v. 5, p. 1, doi. 10.1186/s13395-015-0070-6
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Muscular dystrophy in the mdx mouse is a severe myopathy compounded by hypotrophy, hypertrophy and hyperplasia.
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- Skeletal Muscle, 2015, v. 5, n. 1, p. 1, doi. 10.1186/s13395-015-0041-y
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- Article
Idiopathic inflammatory myopathies: pathogenic mechanisms of muscle weakness.
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- Skeletal Muscle, 2013, v. 3, n. 1, p. 1, doi. 10.1186/2044-5040-3-13
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- Article
Metabolic remodeling agents show beneficial effects in the dystrophin-deficient mdx mouse model.
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- Skeletal Muscle, 2012, v. 2, n. 1, p. 16, doi. 10.1186/2044-5040-2-16
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- Article
Immune-mediated pathology in Duchenne muscular dystrophy.
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- Science Translational Medicine, 2015, v. 7, n. 299, p. 1, doi. 10.1126/scitranslmed.aaa7322
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Biomarker-focused multi-drug combination therapy and repurposing trial in mdx mice.
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- PLoS ONE, 2021, v. 16, n. 2, p. 1, doi. 10.1371/journal.pone.0246507
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Population Pharmacokinetics of Vamorolone (VBP15) in Healthy Men and Boys With Duchenne Muscular Dystrophy.
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- Journal of Clinical Pharmacology, 2019, v. 59, n. 7, p. 979, doi. 10.1002/jcph.1388
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Endoplasmic Reticulum Stress in Skeletal Muscle Homeostasis and Disease.
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- Current Rheumatology Reports, 2012, v. 14, n. 3, p. 238, doi. 10.1007/s11926-012-0247-5
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The TREAT-NMD advisory committee for therapeutics (TACT): an innovative de-risking model to foster orphan drug development.
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- Orphanet Journal of Rare Diseases, 2015, v. 10, n. 1, p. 1, doi. 10.1186/s13023-015-0258-1
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- Article