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G‐quadruplex‐binding small molecules ameliorate <italic>C9orf72 </italic>FTD/ALS pathology <italic>in vitro</italic> and <italic>in vivo</italic>.
Published in:
EMBO Molecular Medicine, 2018, v. 10, n. 1, p. 22, doi. 10.15252/emmm.201707850
By:
- Simone, Roberto;
- Balendra, Rubika;
- Moens, Thomas G.;
- Preza, Elisavet;
- Wilson, Katherine M.;
- Heslegrave, Amanda;
- Woodling, Nathan S.;
- Niccoli, Teresa;
- Gilbert‐Jaramillo, Javier;
- Abdelkarim, Samir;
- Clayton, Emma L.;
- Clarke, Mica;
- Konrad, Marie‐Therese;
- Nicoll, Andrew J.;
- Mitchell, Jamie S.;
- Calvo, Andrea;
- Chio, Adriano;
- Houlden, Henry;
- Polke, James M.;
- Ismail, Mohamed A.
Publication type:
Article
Distinct responses of neurons and astrocytes to TDP-43 proteinopathy in amyotrophic lateral sclerosis.
Published in:
2020
By:
- Smethurst, Phillip;
- Risse, Emmanuel;
- Tyzack, Giulia E;
- Mitchell, Jamie S;
- Taha, Doaa M;
- Chen, Yun-Ru;
- Newcombe, Jia;
- Collinge, John;
- Sidle, Katie;
- Patani, Rickie
Publication type:
journal article
Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosis.
Published in:
Brain: A Journal of Neurology, 2019, v. 142, n. 9, p. 2572, doi. 10.1093/brain/awz217
By:
- Tyzack, Giulia E;
- Luisier, Raphaelle;
- Taha, Doaa M;
- Neeves, Jacob;
- Modic, Miha;
- Mitchell, Jamie S;
- Meyer, Ione;
- Greensmith, Linda;
- Newcombe, Jia;
- Ule, Jernej;
- Luscombe, Nicholas M;
- Patani, Rickie
Publication type:
Article
Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosis.
Published in:
2019
By:
- Tyzack, Giulia E;
- Luisier, Raphaelle;
- Taha, Doaa M;
- Neeves, Jacob;
- Modic, Miha;
- Mitchell, Jamie S;
- Meyer, Ione;
- Greensmith, Linda;
- Newcombe, Jia;
- Ule, Jernej;
- Luscombe, Nicholas M;
- Patani, Rickie
Publication type:
journal article
Sense and antisense RNA are not toxic in <italic>Drosophila</italic> models of <italic>C9orf72</italic>-associated ALS/FTD.
Published in:
Acta Neuropathologica, 2018, v. 135, n. 3, p. 445, doi. 10.1007/s00401-017-1798-3
By:
- Moens, Thomas G.;
- Mizielinska, Sarah;
- Niccoli, Teresa;
- Mitchell, Jamie S.;
- Thoeng, Annora;
- Ridler, Charlotte E.;
- Grönke, Sebastian;
- Esser, Jacqueline;
- Heslegrave, Amanda;
- Zetterberg, Henrik;
- Partridge, Linda;
- Isaacs, Adrian M.
Publication type:
Article
Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS.
Published in:
Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-04373-8
By:
- Luisier, Raphaelle;
- Tyzack, Giulia E.;
- Hall, Claire E.;
- Mitchell, Jamie S.;
- Devine, Helen;
- Taha, Doaa M.;
- Malik, Bilal;
- Meyer, Ione;
- Greensmith, Linda;
- Newcombe, Jia;
- Ule, Jernej;
- Luscombe, Nicholas M.;
- Patani, Rickie
Publication type:
Article

Found: 6

G‐quadruplex‐binding small molecules ameliorate <italic>C9orf72 </italic>FTD/ALS pathology <italic>in vitro</italic> and <italic>in vivo</italic>.

Distinct responses of neurons and astrocytes to TDP-43 proteinopathy in amyotrophic lateral sclerosis.

Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosis.

Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosis.

Sense and antisense RNA are not toxic in <italic>Drosophila</italic> models of <italic>C9orf72</italic>-associated ALS/FTD.

Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS.