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Synthesis and Biological in vitro and in vivo Evaluation of 2‐(5‐Nitroindazol‐1‐yl)ethylamines and Related Compounds as Potential Therapeutic Alternatives for Chagas Disease.
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- ChemMedChem, 2018, v. 13, n. 19, p. 2104, doi. 10.1002/cmdc.201800512
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Site-to-site interdomain communication may mediate different loss-of-function mechanisms in a cancer-associated NQO1 polymorphism.
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- Scientific Reports, 2017, p. 44532, doi. 10.1038/srep44532
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Erratum: Conformational dynamics is key to understanding loss-of-function of NQO1 cancer-associated polymorphisms and its correction by pharmacological ligands.
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- Scientific Reports, 2016, p. 21939, doi. 10.1038/srep21939
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Conformational dynamics is key to understanding loss-of-function of NQO1 cancer-associated polymorphisms and its correction by pharmacological ligands.
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- Scientific Reports, 2016, p. 20331, doi. 10.1038/srep20331
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Cell Survival Enabled by Leakage of a Labile Metabolic Intermediate.
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- Molecular Biology & Evolution, 2023, v. 40, n. 3, p. 1, doi. 10.1093/molbev/msad032
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Insight into the specificity and severity of pathogenic mechanisms associated with missense mutations through experimental and structural perturbation analyses.
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- Human Molecular Genetics, 2019, v. 28, n. 1, p. 1, doi. 10.1093/hmg/ddy323
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Enhanced vulnerability of human proteins towards disease-associated inactivation through divergent evolution.
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- Human Molecular Genetics, 2017, v. 26, n. 18, p. 3531, doi. 10.1093/hmg/ddx238
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A mechanism for cancer-associated inactivation of NQO1 due to P187S and its reactivation by the consensus mutation H80R.
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- FEBS Letters, 2017, v. 591, n. 18, p. 2826, doi. 10.1002/1873-3468.12772
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