Works by Meagher, Craig
Results: 8
Response to Comment on: Meagher et al. Neutralization of Interleukin-16 Protects Nonobese Diabetic Mice From Autoimmune Type 1 Diabetes by a CCL4-Dependent Mechanism. Diabetes 2010;59:2862–2871.
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- Diabetes, 2011, v. 60, n. 2, p. e13, doi. 10.2337/db10-1620
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- Article
Neutralization of Interleukin-16 Protects Nonobese Diabetic Mice From Autoimmune Type 1 Diabetes by a CCL4-Dependent Mechanism.
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- Diabetes, 2010, v. 59, n. 11, p. 2862, doi. 10.2337/db09-0131
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- Article
CCL4 protects from type 1 diabetes by altering islet beta-cell-targeted inflammatory responses.
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- 2007
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- journal article
CCL4 Protects From Type 1 Diabetes by Altering Islet β-Cell-Targeted Inflammatory Responses.
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- Diabetes, 2007, v. 56, n. 3, p. 809, doi. 10.2337/db06-0619
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- Article
Blockade of tumor necrosis factor-related apoptosis-inducing ligand exacerbates type 1 diabetes in NOD mice.
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- 2003
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- journal article
Congenic mapping of the diabetogenic locus Idd4 to a 5.2-cM region of chromosome 11 in NOD mice: identification of two potential candidate subloci.
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- 2002
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- journal article
Translational findings for odronextamab: From preclinical research to a first‐in‐human study in patients with CD20+ B‐cell malignancies.
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- CTS: Clinical & Translational Science, 2022, v. 15, n. 4, p. 954, doi. 10.1111/cts.13212
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- Article
NK Cells Are Not Required for Spontaneous Autoimmune Diabetes in NOD Mice.
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- PLoS ONE, 2012, v. 7, n. 4, p. 1, doi. 10.1371/journal.pone.0036011
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- Article