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Biogenesis of P-TEFb in CD4<sup>+</sup> T cells to reverse HIV latency is mediated by protein kinase C (PKC)-independent signaling pathways.
- Published in:
- PLoS Pathogens, 2021, v. 17, n. 9, p. 1, doi. 10.1371/journal.ppat.1009581
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- Article
Phosphorylation of HEXIM1 at Tyr271 and Tyr274 Promotes Release of P-TEFb from the 7SK snRNP Complex and Enhances Proviral HIV Gene Expression.
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- Proteomics, 2015, v. 15, n. 12, p. 2078, doi. 10.1002/pmic.201500038
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- Article
Exosomes derived from HIV-1-infected cells promote growth and progression of cancer via HIV TAR RNA.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-07006-2
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- Article
The KAT5-Acetyl-Histone4-Brd4 axis silences HIV-1 transcription and promotes viral latency.
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- PLoS Pathogens, 2018, v. 14, n. 3, p. 1, doi. 10.1371/journal.ppat.1007012
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- Article
The cell biology of HIV-1 latency and rebound.
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- Retrovirology, 2024, v. 21, n. 1, p. 1, doi. 10.1186/s12977-024-00639-w
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- Article
Phosphorylation of CDK9 at Ser175 Enhances HIV Transcription and Is a Marker of Activated P-TEFb in CD4<sup>+</sup> T Lymphocytes
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- PLoS Pathogens, 2013, v. 9, n. 5, p. 1, doi. 10.1371/journal.ppat.1003338
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- Article