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The matricellular protein CCN1 enhances TGF-β1/SMAD3-dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to lung injury.
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- FASEB Journal, 2016, v. 30, n. 6, p. 2135, doi. 10.1096/fj.201500173
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- Article
Impaired Myofibroblast Dedifferentiation Contributes to Nonresolving Fibrosis in Aging.
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- American Journal of Respiratory Cell & Molecular Biology, 2020, v. 62, n. 5, p. 633, doi. 10.1165/rcmb.2019-0092OC
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- Article
Novel Mechanisms for the Antifibrotic Action of Nintedanib.
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- American Journal of Respiratory Cell & Molecular Biology, 2016, v. 54, n. 1, p. 51, doi. 10.1165/rcmb.2014-0445OC
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- Article
Novel Mechanisms for the Anti-Fibrotic Action of Nintedanib.
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- American Journal of Respiratory Cell & Molecular Biology, 2016, v. 54, n. 1, p. E1, doi. 10.1165/rcmb.2014-0445oc
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- Article
Metformin, an Antidiabetic Agent Reduces Growth of Cutaneous Squamous Cell Carcinoma by Targeting mTOR Signaling Pathway<sup>†</sup> Metformin, an Antidiabetic Agent Reduces Growth of Cutaneous Squamous Cell Carcinoma by Targeting mTOR Signaling Pathway
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- Photochemistry & Photobiology, 2012, v. 88, n. 5, p. 1149, doi. 10.1111/j.1751-1097.2012.01165.x
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- Article
Reversal of Persistent Fibrosis in Aging by Targeting Nox4-Nrf2 Redox Imbalance.
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- Science Translational Medicine, 2014, v. 6, n. 231, p. 1, doi. 10.1126/scitranslmed.3008182
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- Article
Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental pulmonary fibrosis.
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- Journal of Clinical Investigation, 2013, v. 123, n. 3, p. 1096, doi. 10.1172/JCI66700
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- Article
Mitochondrial uncoupling protein‐2 reprograms metabolism to induce oxidative stress and myofibroblast senescence in age‐associated lung fibrosis.
- Published in:
- Aging Cell, 2022, v. 21, n. 9, p. 1, doi. 10.1111/acel.13674
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- Article