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Genetic Deletion of Polo-Like Kinase 2 Induces a Pro-Fibrotic Pulmonary Phenotype.
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- Cells (2073-4409), 2021, v. 10, n. 3, p. 617, doi. 10.3390/cells10030617
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- Article
Targeting cardiomyocyte ADAM10 ectodomain shedding promotes survival early after myocardial infarction.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-022-35331-0
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- Article
The reduced activity of PP-1α under redox stress condition is a consequence of GSH-mediated transient disulfide formation.
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- Scientific Reports, 2018, v. 8, n. 1, p. 1, doi. 10.1038/s41598-018-36267-6
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- Article
Cellular Mechanisms of the Anti-Arrhythmic Effect of Cardiac PDE2 Overexpression.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 9, p. 4816, doi. 10.3390/ijms22094816
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- Article
Therapeutic Implications for PDE2 and cGMP/cAMP Mediated Crosstalk in Cardiovascular Diseases.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 20, p. 7462, doi. 10.3390/ijms21207462
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- Article
Intertwined regulators: hypoxia pathway proteins, microRNAs, and phosphodiesterases in the control of steroidogenesis.
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- Pflügers Archiv: European Journal of Physiology, 2024, v. 476, n. 9, p. 1383, doi. 10.1007/s00424-024-02921-4
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- Article
Investigation of mesalazine as an antifibrotic drug following myocardial infarction in male mice.
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- Physiological Reports, 2023, v. 11, n. 17, p. 1, doi. 10.14814/phy2.15809
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- Article
Gene therapy with phosphodiesterases 2A and 4B ameliorates heart failure and arrhythmias by improving subcellular cAMP compartmentation.
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- Cardiovascular Research, 2024, v. 120, n. 9, p. 1011, doi. 10.1093/cvr/cvae094
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- Article