Found: 19
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Role of tropomyosin isoforms in the calcium sensitivity of striated muscle thin filaments.
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- Journal of Muscle Research & Cell Motility, 2007, v. 28, n. 1, p. 49
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- Article
Enhanced Cardiomyocyte Function in Hypertensive Rats With Diastolic Dysfunction and Human Heart Failure Patients After Acute Treatment With Soluble Guanylyl Cyclase (sGC) Activator.
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- Frontiers in Physiology, 2020, v. 11, p. 1, doi. 10.3389/fphys.2020.00345
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- Article
Modulation of Titin-Based Stiffness in Hypertrophic Cardiomyopathy via Protein Kinase D.
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- Frontiers in Physiology, 2020, p. 1, doi. 10.3389/fphys.2020.00240
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- Article
Functional Characterization of Cardiac Actin Mutants Causing Hypertrophic (p.A295S) and Dilated Cardiomyopathy (p.R312H and p.E361G).
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- International Journal of Molecular Sciences, 2022, v. 23, n. 8, p. 4465, doi. 10.3390/ijms23084465
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- Article
Genetic Restrictive Cardiomyopathy: Causes and Consequences—An Integrative Approach.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 2, p. 558, doi. 10.3390/ijms22020558
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- Article
Cardiac troponin C-L29Q, related to hypertrophic cardiomyopathy, hinders the transduction of the protein kinase A dependent phosphorylation signal from cardiac troponin I to C.
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- FEBS Journal, 2005, v. 272, n. 23, p. 6087, doi. 10.1111/j.1742-4658.2005.05001.x
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- Article
PKCα-Specific Phosphorylation of the Troponin Complex in Human Myocardium: A Functional and Proteomics Analysis.
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- PLoS ONE, 2013, v. 8, n. 10, p. 1, doi. 10.1371/journal.pone.0074847
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- Article
Myofilament protein carbonylation contributes to the contractile dysfunction in the infarcted LV region of mouse hearts.
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- Cardiovascular Research, 2014, v. 101, n. 1, p. 108, doi. 10.1093/cvr/cvt236
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- Article
Functional effects of protein kinase C-mediated myofilament phosphorylation in human myocardium
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- Cardiovascular Research, 2006, v. 69, n. 4, p. 876, doi. 10.1016/j.cardiores.2005.11.021
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- Article
Expression of cTnI-R145G affects shortening properties of adult rat cardiomyocytes.
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- Pflügers Archiv: European Journal of Physiology, 2008, v. 457, n. 1, p. 17, doi. 10.1007/s00424-008-0487-4
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- Article
Sex-specific cardiovascular remodeling leads to a divergent sex-dependent development of heart failure in aged hypertensive rats.
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- GeroScience, 2024, v. 46, n. 5, p. 4543, doi. 10.1007/s11357-024-01160-w
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- Article
Infantile restrictive cardiomyopathy: cTnI-R170G/W impair the interplay of sarcomeric proteins and the integrity of thin filaments.
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- PLoS ONE, 2020, v. 15, n. 3, p. 1, doi. 10.1371/journal.pone.0229227
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- Article
Secretory products of guinea pig epicardial fat induce insulin resistance and impair primary adult rat cardiomyocyte function.
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- Journal of Cellular & Molecular Medicine, 2011, v. 15, n. 11, p. 2399, doi. 10.1111/j.1582-4934.2010.01232.x
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- Article
The Interplay between S-Glutathionylation and Phosphorylation of Cardiac Troponin I and Myosin Binding Protein C in End-Stage Human Failing Hearts.
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- Antioxidants, 2021, v. 10, n. 7, p. 1134, doi. 10.3390/antiox10071134
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- Article
Integration of Cardiac Actin Mutants Causing Hypertrophic (p.A295S) and Dilated Cardiomyopathy (p.R312H and p.E361G) into Cellular Structures.
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- Antioxidants, 2021, v. 10, n. 7, p. 1082, doi. 10.3390/antiox10071082
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- Article
The interaction of the bisphosphorylated N-terminal arm of cardiac troponin I-A <sup>31</sup>P-NMR study
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- FEBS Letters, 2002, v. 513, n. 2/3, p. 289, doi. 10.1016/S0014-5793(02)02340-2
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- Article
Protein Kinase D Plays a Crucial Role in Maintaining Cardiac Homeostasis by Regulating Post-Translational Modifications of Myofilament Proteins.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 5, p. 2790, doi. 10.3390/ijms25052790
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- Article
Protein kinase C α and ε phosphorylation of troponin and myosin binding protein C reduce Ca<sup>2+</sup> sensitivity in human myocardium.
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- Basic Research in Cardiology, 2010, v. 105, n. 2, p. 289, doi. 10.1007/s00395-009-0053-z
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- Article
Soluble adenylyl cyclase: A novel player in cardiac hypertrophy induced by isoprenaline or pressure overload.
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- PLoS ONE, 2018, v. 13, n. 2, p. 1, doi. 10.1371/journal.pone.0192322
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- Article