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Divergent downstream biosynthetic pathways are supported by L-cysteine synthases of Mycobacterium tuberculosis.
- Published in:
- eLife, 2024, p. 1, doi. 10.7554/eLife.91970
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- Article
Structure of a Wbl protein and implications for NO sensing by M. tuberculosis.
- Published in:
- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-02418-y
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- Article
Spectrum of dominant mutations in the desmosomal cadherin desmoglein 1, causing the skin disease striate palmoplantar keratoderma.
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- European Journal of Human Genetics, 2001, v. 9, n. 3, p. 197, doi. 10.1038/sj.ejhg.5200605
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- Article
Flexible nitrogen utilisation by the metabolic generalist pathogen Mycobacterium tuberculosis.
- Published in:
- eLife, 2019, p. 1, doi. 10.7554/eLife.41129
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- Article
An Intramolecular Switch Regulates Phosphoindependent FHA Domain Interactions in Mycobacterium tuberculosis.
- Published in:
- Science Signaling, 2009, v. 2, n. 63, p. 1, doi. 10.1126/scisignal.2000212
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- Article
Cmr is a redox-responsive regulator of DosR that contributes to M. tuberculosis virulence.
- Published in:
- Nucleic Acids Research, 2017, v. 45, n. 11, p. 6600, doi. 10.1093/nar/gkx406
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- Article
Genomic mapping of cAMP receptor protein (CRPMt) in Mycobacterium tuberculosis: relation to transcriptional start sites and the role of CRPMt as a transcription factor.
- Published in:
- Nucleic Acids Research, 2014, v. 42, n. 13, p. 8320, doi. 10.1093/nar/gku548
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- Article
A member of the cAMP receptor protein family of transcription regulators in Mycobacterium tuberculosis is required for virulence in mice and controls transcription of the rpfA gene coding for a resuscitation promoting factor.
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- 2005
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- Correction notice
A member of the cAMP receptor protein family of transcription regulators inMycobacterium tuberculosisis required for virulence in mice and controls transcription of therpfAgene coding for a resuscitation promoting factor.
- Published in:
- Molecular Microbiology, 2005, v. 56, n. 5, p. 1274, doi. 10.1111/j.1365-2958.2005.04609.x
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- Article
N-terminal deletion in a desmosomal cadherin causes the autosomal dominant skin disease striate palmoplantar keratoderma.
- Published in:
- Human Molecular Genetics, 1999, v. 8, n. 6, p. 971, doi. 10.1093/hmg/8.6.971
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- Article