Found: 9
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Membralin deficiency dysregulates astrocytic glutamate homeostasis leading to ALS-like impairment.
- Published in:
- 2019
- By:
- Publication type:
- journal article
Appoptosin Mediates Lesions Induced by Oxidative Stress Through the JNK-FoxO1 Pathway.
- Published in:
- Frontiers in Aging Neuroscience, 2019, p. 1, doi. 10.3389/fnagi.2019.00243
- By:
- Publication type:
- Article
Soluble SORLA Enhances Neurite Outgrowth and Regeneration through Activation of the EGF Receptor/ERK Signaling Axis.
- Published in:
- Journal of Neuroscience, 2020, v. 40, n. 31, p. 5908, doi. 10.1523/jneurosci.0723-20.2020
- By:
- Publication type:
- Article
Amyloid, tau, pathogen infection and antimicrobial protection in Alzheimer’s disease –conformist, nonconformist, and realistic prospects for AD pathogenesis.
- Published in:
- Translational Neurodegeneration, 2018, v. 7, n. 1, p. 1, doi. 10.1186/s40035-018-0139-3
- By:
- Publication type:
- Article
SNX17 Mediates Dendritic Spine Maturation via p140Cap.
- Published in:
- Molecular Neurobiology, 2024, v. 61, n. 3, p. 1346, doi. 10.1007/s12035-023-03620-4
- By:
- Publication type:
- Article
Trem2 deletion enhances tau dispersion and pathology through microglia exosomes.
- Published in:
- Molecular Neurodegeneration, 2022, v. 17, n. 1, p. 1, doi. 10.1186/s13024-022-00562-8
- By:
- Publication type:
- Article
LILRB2-mediated TREM2 signaling inhibition suppresses microglia functions.
- Published in:
- Molecular Neurodegeneration, 2022, v. 17, n. 1, p. 1, doi. 10.1186/s13024-022-00550-y
- By:
- Publication type:
- Article
Molecular and cellular mechanisms underlying the pathogenesis of Alzheimer's disease.
- Published in:
- Molecular Neurodegeneration, 2020, v. 15, n. 1, p. 1, doi. 10.1186/s13024-020-00391-7
- By:
- Publication type:
- Article
SNX27 and SORLA Interact to Reduce Amyloidogenic Subcellular Distribution and Processing of Amyloid Precursor Protein.
- Published in:
- Journal of Neuroscience, 2016, v. 36, n. 30, p. 7996, doi. 10.1523/JNEUROSCI.0206-16.2016
- By:
- Publication type:
- Article