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DNA damage in embryonic neural stem cell determines FTLDs’ fate via early-stage neuronal necrosis.
- Published in:
- Life Science Alliance, 2021, v. 4, n. 5, p. 1, doi. 10.26508/lsa.202101022
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- Article
Fasting activates macroautophagy in neurons of Alzheimer's disease mouse model but is insufficient to degrade amyloid-beta.
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- Scientific Reports, 2015, p. 12115, doi. 10.1038/srep12115
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- Article
PQBP5/NOL10 maintains and anchors the nucleolus under physiological and osmotic stress conditions.
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- Nature Communications, 2023, v. 14, n. 1, p. 1, doi. 10.1038/s41467-022-35602-w
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- Article
Comprehensive phosphoproteome analysis unravels the core signaling network that initiates the earliest synapse pathology in preclinical Alzheimer’s disease brain.
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- Human Molecular Genetics, 2015, v. 24, n. 2, p. 540, doi. 10.1093/hmg/ddu475
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- Article
Targeting Tyro3 ameliorates a model of PGRNmutant FTLD-TDP via tau-mediated synaptic pathology.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-02821-z
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- Article
Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1.
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- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-01790-z
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- Article
Necrosis Links Neurodegeneration and Neuroinflammation in Neurodegenerative Disease.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 7, p. 3636, doi. 10.3390/ijms25073636
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- Article
HMGB1 facilitates repair of mitochondrial DNA damage and extends the lifespan of mutant ataxin-1 knock-in mice.
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- EMBO Molecular Medicine, 2015, v. 7, n. 1, p. 78, doi. 10.15252/emmm.201404392
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- Article
Identification of hepta-histidine as a candidate drug for Huntington's disease by in silico-in vitro- in vivo-integrated screens of chemical libraries.
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- Scientific Reports, 2016, p. 33861, doi. 10.1038/srep33861
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- Article
HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer's disease.
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- Scientific Reports, 2016, p. 31895, doi. 10.1038/srep31895
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- Article
Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation.
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- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-26851-2
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- Article
HMGB1 signaling phosphorylates Ku70 and impairs DNA damage repair in Alzheimer's disease pathology.
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- Communications Biology, 2021, v. 4, n. 1, p. 1, doi. 10.1038/s42003-021-02671-4
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- Article
YAP-dependent necrosis occurs in early stages of Alzheimer's disease and regulates mouse model pathology.
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- Nature Communications, 2020, v. 11, n. 1, p. 1, doi. 10.1038/s41467-020-14353-6
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- Article
Targeting TEAD/YAP-transcription-dependent necrosis, TRIAD, ameliorates Huntington’s disease pathology.
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- Human Molecular Genetics, 2016, v. 25, n. 21, p. 4749, doi. 10.1093/hmg/ddw303
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- Article
RpA1 ameliorates symptoms of mutant ataxin-1 knock-in mice and enhances DNA damage repair.
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- Human Molecular Genetics, 2016, v. 25, n. 20, p. 4432, doi. 10.1093/hmg/ddw272
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- Article
Dynamic molecular network analysis of iPSC-Purkinje cells differentiation delineates roles of ISG15 in SCA1 at the earliest stage.
- Published in:
- Communications Biology, 2024, v. 7, n. 1, p. 1, doi. 10.1038/s42003-024-06066-z
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- Article