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The Volume Activated Potassium Channel KCNK5 is Up-Regulated in Activated Human T Cells, but Volume Regulation is Impaired.
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- Cellular Physiology & Biochemistry (Karger AG), 2016, v. 38, n. 5, p. 883, doi. 10.1159/000443042
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The Volume Activated Potassium Channel KCNK5 is Up-Regulated in Activated Human T Cells, but Volume Regulation is Impaired.
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- Cellular Physiology & Biochemistry (Karger AG), 2016, v. 38, n. 3, p. 883, doi. 10.1159/000443042
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CrossTalk proposal: Cell volume changes are an essential step in the cell death machinery.
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- Journal of Physiology, 2013, v. 591, n. 24, p. 6119, doi. 10.1113/jphysiol.2013.258632
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Rebuttal from Florian Lang and Else K. Hoffmann.
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- Journal of Physiology, 2013, v. 591, n. 24, p. 6127, doi. 10.1113/jphysiol.2013.265231
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Cell volume regulation in epithelial physiology and cancer.
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- Frontiers in Physiology, 2013, v. 3, p. 1
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Double Mutation at the Putative Protein Kinase C Phosphorylation Sites Thr<sup>151</sup> Plus Thr<sup>323</sup> in the Mouse LeukotrieneD<sub>4</sub> Receptor Eliminates Homologous Desensitization.
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- Cellular Physiology & Biochemistry (Karger AG), 2013, v. 31, n. 2/3, p. 366, doi. 10.1159/000343374
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Cell Volume Regulation and Signaling in 3T3-L1 Pre-adipocytes and Adipocytes: On the Possible Roles of Caveolae, Insulin Receptors, FAK and ERK1/2.
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- Cellular Physiology & Biochemistry (Karger AG), 2011, v. 28, n. 6, p. 1231, doi. 10.1159/000335855
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Differential Role for ERK2 in Anoxia-induced Activation of Transcription and Translation of Hsp70 in NIH 3T3 Cells.
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- Cellular Physiology & Biochemistry (Karger AG), 2011, v. 27, n. 2, p. 109, doi. 10.1159/000325213
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Pinpointing Differences in Cisplatin-induced Apoptosis in Adherent and Non-adherent Cancer Cells.
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- Cellular Physiology & Biochemistry (Karger AG), 2010, v. 26, n. 6, p. 809, doi. 10.1159/000323990
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Directional Cell Migration and Chemotaxis in Wound Healing Response to PDGF-AA are Coordinated by the Primary Cilium in Fibroblasts.
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- Cellular Physiology & Biochemistry (Karger AG), 2010, v. 25, n. 2/3, p. 279, doi. 10.1159/000276562
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Comparison of two anoxia models in rainbow trout cells by a 2-DE and MS/MS-based proteome approach.
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- Proteomics, 2008, v. 8, n. 10, p. 2035, doi. 10.1002/pmic.200700944
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Molecular and Functional Expression of High Conductance Ca Activated K<sup>+</sup> Channels in the Eel Intestinal Epithelium.
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- Cellular Physiology & Biochemistry (Karger AG), 2008, v. 21, n. 5/6, p. 373, doi. 10.1159/000129630
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H-ras transformation sensitizes volume-activated anion channels and increases migratory activity of NIH3T3 fibroblasts.
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- Pflügers Archiv: European Journal of Physiology, 2008, v. 455, n. 6, p. 1055, doi. 10.1007/s00424-007-0367-3
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Long term anoxia in rainbow trout investigated by 2-DE and MS/MS.
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- Proteomics, 2008, v. 8, n. 5, p. 1009, doi. 10.1002/pmic.200700460
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- Article
Homologous Desensitisation of the Mouse Leukotriene B<sub>4</sub> Receptor Involves Protein Kinase C-Mediated Phosphorylation of Serine 127.
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- Cellular Physiology & Biochemistry (Karger AG), 2007, v. 20, n. 1-4, p. 143, doi. 10.1159/000104162
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Cell cycle-dependent activity of the volume- and Ca<sup>2+</sup>-activated anion currents in Ehrlich lettre ascites cells.
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- Journal of Cellular Physiology, 2007, v. 210, n. 3, p. 831, doi. 10.1002/jcp.20918
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Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.
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- Journal of Physiology, 2005, v. 567, n. 2, p. 427, doi. 10.1113/jphysiol.2005.087130
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A cell shrinkage-induced non-selective cation conductance with a novel pharmacology in Ehrlich–Lettre-ascites tumour cells
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- FEBS Letters, 2003, v. 539, n. 1-3, p. 115, doi. 10.1016/S0014-5793(03)00210-2
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- Article
Rho family GTP binding proteins are involved in the regulatory volume decrease process in NIH3T3 mouse fibroblasts.
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- Journal of Physiology, 2002, v. 541, n. 3, p. 779, doi. 10.1113/jphysiol.2002.018887
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- Article
Regulation of the Na+2Cl−K+ co‐transporter—mechanisms in the rectal gland of Squalus acanthias with implications for the thick ascending limb of Henle.
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- Nephrology Dialysis Transplantation, 2000, v. 15, p. 16, doi. 10.1093/ndt/15.suppl_6.16
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Intracellular Signalling Involved in Volume Regulatory Decrease.
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- Cellular Physiology & Biochemistry (Karger AG), 2000, v. 10, n. 5/6, p. 273, doi. 10.1159/000016356
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K<sup>+</sup> currents activated by leukotriene D<sub>4</sub> or osmotic swelling in Ehrlich ascites tumour cells.
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- Pflügers Archiv: European Journal of Physiology, 2000, v. 440, n. 2, p. 283, doi. 10.1007/s004240000273
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Characterisation of a cell swelling-activated K<sup>+</sup>-selective conductance of Ehrlich mouse ascites tumour cells.
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- Journal of Physiology, 2000, v. 524, n. 3, p. 757, doi. 10.1111/j.1469-7793.2000.00757.x
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pH Regulation in Sensitive and Multidrug Resistant Ehrlich Ascites Tumor Cells.
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- Cellular Physiology & Biochemistry (Karger AG), 1998, v. 8, n. 3, p. 138, doi. 10.1159/000016277
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Mechanisms of pH<sub>i</sub> regulation studied in individual neurons cultured from mouse cerebral cortex.
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- Journal of Neuroscience Research, 1998, v. 51, n. 4, p. 431, doi. 10.1002/(SICI)1097-4547(19980215)51:4<431::AID-JNR3>3.0.CO;2-D
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Antiserum against Proteins of the Na<sup>+</sup>, K<sup>+</sup>,2CI<sup>-</sup> Cotransporter in Ehrlich Ascites Tumor Cells Inhibits Volume Regulation and Bumetanide-Sensitive K<sup>+</sup> Influx.
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- Cellular Physiology & Biochemistry (Karger AG), 1995, v. 5, n. 2, p. 107, doi. 10.1159/000154745
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HgCI<sub>2</sub>-lnduced Ion Transport Pathways in Ehrlich Ascites Tumor Cells.
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- Cellular Physiology & Biochemistry (Karger AG), 1993, v. 3, n. 2, p. 97, doi. 10.1159/000154672
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Role of Amino Acid Transport System A in the Control of Cell Volume in Cultured Human Fibroblasts.
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- Cellular Physiology & Biochemistry (Karger AG), 1991, v. 1, n. 3, p. 131, doi. 10.1159/000154601
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Cell volume regulation in ehrlich ascites tumor cells.
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- Journal of Cellular Physiology, 1974, v. 84, n. 1, p. 115, doi. 10.1002/jcp.1040840113
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