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Per-1 is a specific clock gene regulated by parathyroid hormone (PTH) signaling in osteoblasts and is functional for the transcriptional events induced by PTH.
- Published in:
- Journal of Cellular Biochemistry, 2011, v. 112, n. 2, p. 433, doi. 10.1002/jcb.22957
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- Article
Osteoclast-specific Dicer gene deficiency suppresses osteoclastic bone resorption.
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- Journal of Cellular Biochemistry, 2010, v. 109, n. 5, p. 866, doi. 10.1002/jcb.22228
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- Article
Conventional Type 1 Dendritic Cells in Intestinal Immune Homeostasis.
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- Frontiers in Immunology, 2022, v. 13, p. 1, doi. 10.3389/fimmu.2022.857954
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- Article
IκB kinase-α is critical for interferon-α production induced by Toll-like receptors 7 and 9.
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- Nature, 2006, v. 440, n. 7086, p. 949, doi. 10.1038/nature04641
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- Article
Regulation of Toll/IL-1-receptor-mediated gene expression by the inducible nuclear protein I?B?
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- Nature, 2004, v. 430, n. 6996, p. 218, doi. 10.1038/nature02738
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- Article
Essential role for TIRAP in activation of the signalling cascade shared by TLR2 and TLR4.
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- Nature, 2002, v. 420, n. 6913, p. 324, doi. 10.1038/nature01182
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- Article
Hindlimb-unloading suppresses B cell population in the bone marrow and peripheral circulation associated with OPN expression in circulating blood cells.
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- Journal of Bone & Mineral Metabolism, 2015, v. 33, n. 1, p. 48, doi. 10.1007/s00774-014-0568-8
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- Article
Augmentation of Stimulator of Interferon Genes–Induced Type I Interferon Production in COPA Syndrome.
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- Arthritis & Rheumatology, 2021, v. 73, n. 11, p. 2105, doi. 10.1002/art.41790
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- Article
Interferon response induced by Toll-like receptor signaling.
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- Innate Immunity, 2004, v. 10, n. 4, p. 252, doi. 10.1177/09680519040100040901
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- Article
An IFN-γ-IL-18 Signaling Loop Accelerates Memory CD8<sup>+</sup> T Cell Proliferation.
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- PLoS ONE, 2008, v. 3, n. 6, p. 1, doi. 10.1371/journal.pone.0002404
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- Article
Differential Contribution of Osteoclast- and Osteoblast-Lineage Cells to CpG-Oligodeoxynucleotide (CpG-ODN) Modulation of Osteoclastogenesis.
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- Journal of Bone & Mineral Research, 2005, v. 20, n. 9, p. 1692, doi. 10.1359/JBMR.050515
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- Article
Innate Vα14<sup>+</sup> natural killer T cells mature dendritic cells, leading to strong adaptive immunity.
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- Immunological Reviews, 2007, v. 220, n. 1, p. 183, doi. 10.1111/j.1600-065X.2007.00561.x
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- Article
Crucial roles of XCR1-expressing dendritic cells and the XCR1-XCL1 chemokine axis in intestinal immune homeostasis.
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- Scientific Reports, 2016, p. 23505, doi. 10.1038/srep23505
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- Article
Cholera toxin B induces interleukin-1β production from resident peritoneal macrophages through the pyrin inflammasome as well as the NLRP3 inflammasome.
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- International Immunology, 2019, v. 31, n. 10, p. 657, doi. 10.1093/intimm/dxz004
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- Article
Heterozygous missense variant of the proteasome subunit β-type 9 causes neonatal-onset autoinflammation and immunodeficiency.
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- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-27085-y
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- Article
A Toll-like receptor recognizes bacterial DNA.
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- Nature, 2000, v. 408, n. 6813, p. 740, doi. 10.1038/35047123
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- Article
The Ets transcription factor Spi-B is essential for the differentiation of intestinal microfold cells.
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- Nature Immunology, 2012, v. 13, n. 8, p. 729, doi. 10.1038/ni.2352
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- Publication type:
- Article
Detection of pathogenic intestinal bacteria by Toll-like receptor 5 on intestinal CD11c<sup>+</sup> lamina propria cells.
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- Nature Immunology, 2006, v. 7, n. 8, p. 868, doi. 10.1038/ni1362
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- Article
Erratum: A Toll-like receptor–independent antiviral response induced by double-stranded B-form DNA.
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- 2006
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- Correction notice
A Toll-like receptor–independent antiviral response induced by double-stranded B-form DNA.
- Published in:
- Nature Immunology, 2006, v. 7, n. 1, p. 40, doi. 10.1038/ni1282
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- Publication type:
- Article
Interferon-ainduction through Toll-like receptors involves a direct interaction of IRF7 with MyD88 and TRAF6.
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- Nature Immunology, 2004, v. 5, n. 10, p. 1061, doi. 10.1038/ni1118
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- Publication type:
- Article
TRAM is specifically involved in the Toll-like receptor 4-mediated MyD88-independent signaling pathway.
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- Nature Immunology, 2003, v. 4, n. 11, p. 1144, doi. 10.1038/ni986
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- Article
Small anti-viral compounds activate immune cells via the TLR7 MyD88?dependent signaling pathway.
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- Nature Immunology, 2002, v. 3, n. 2, p. 196, doi. 10.1038/ni758
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- Article
Identification of a novel CCDC22 mutation in a patient with severe Epstein-Barr virus-associated hemophagocytic lymphohistiocytosis and aggressive natural killer cell leukemia.
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- 2019
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- Publication type:
- journal article
Osteoblastic bone formation is induced by using nanogel-crosslinking hydrogel as novel scaffold for bone growth factor.
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- Journal of Cellular Physiology, 2009, v. 220, n. 1, p. 1, doi. 10.1002/jcp.21760
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- Article
CpG-activated Thy1.2.
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- European Journal of Immunology, 2005, v. 35, n. 8, p. 2397, doi. 10.1002/eji.200425795
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- Article
The Toll-like receptor 7 (TLR7)-specific stimulus loxoribine uncovers a strong relationship within the TLR7, 8 and 9 subfamily.
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- European Journal of Immunology, 2003, v. 33, n. 11, p. 2987, doi. 10.1002/eji.200324238
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- Article
CIZ/NMP4 is expressed in B16 melanoma and forms a positive feedback loop with RANKL to promote migration of the melanoma cells.
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- Journal of Cellular Physiology, 2012, v. 227, n. 7, p. 2807, doi. 10.1002/jcp.24066
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- Article
Dok-1 and Dok-2 deficiency induces osteopenia via activation of osteoclasts.
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- Journal of Cellular Physiology, 2011, v. 226, n. 12, p. 3087, doi. 10.1002/jcp.22909
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- Article
Schnurri-2 deficiency counteracts against bone loss induced by ovariectomy.
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- Journal of Cellular Physiology, 2011, v. 226, n. 3, p. 573, doi. 10.1002/jcp.22521
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- Article
Anticancer effects of chemokine-directed antigen delivery to a cross-presenting dendritic cell subset with immune checkpoint blockade.
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- 2020
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- Publication type:
- journal article
Programmed cell death ligand 1 disruption by clustered regularly interspaced short palindromic repeats/Cas9‐genome editing promotes antitumor immunity and suppresses ovarian cancer progression.
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- Cancer Science, 2019, v. 110, n. 4, p. 1279, doi. 10.1111/cas.13958
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- Article