Found: 10
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Diabetic conditions downregulate the expression of CD2AP in podocytes via PI3-K/Akt signalling.
- Published in:
- Diabetes/Metabolism Research & Reviews, 2015, v. 31, n. 1, p. 50, doi. 10.1002/dmrr.2562
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- Publication type:
- Article
X-linked recessive nephrogenic diabetes insipidus: a clinico-genetic study.
- Published in:
- Journal of Pediatric Endocrinology & Metabolism, 2014, v. 27, n. 1/2, p. 93, doi. 10.1515/jpem-2013-0231
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- Publication type:
- Article
Matrix Metalloproteinases and Bullae in Pediatric Henoch Schönlein Purpura.
- Published in:
- 2011
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- Publication type:
- Letter
Beta-catenin and its alteration in an experimental model of diabetic nephropathy.
- Published in:
- 2014
- By:
- Publication type:
- Journal Article
The role of tumor necrosis factor-a in Henoch-Schönlein purpura.
- Published in:
- Pediatric Nephrology, 2005, v. 20, n. 2, p. 149, doi. 10.1007/s00467-004-1726-3
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- Publication type:
- Article
Cerebral vasculitis in Henoch-Schönlein purpura: a case report with sequential magnetic resonance imaging.
- Published in:
- Pediatric Nephrology, 1996, v. 10, n. 5, p. 634, doi. 10.1007/s004670050177
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- Publication type:
- Article
A nationwide study of mass urine screening tests on Korean school children and implications for chronic kidney disease management.
- Published in:
- Clinical & Experimental Nephrology, 2013, v. 17, n. 2, p. 205, doi. 10.1007/s10157-012-0672-9
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- Publication type:
- Article
Angiotensin II suppresses adenosine monophosphate-activated protein kinase of podocytes via angiotensin II type 1 receptor and mitogen-activated protein kinase signaling.
- Published in:
- Clinical & Experimental Nephrology, 2013, v. 17, n. 1, p. 16, doi. 10.1007/s10157-012-0649-8
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- Publication type:
- Article
Montelukast improves the changes of cytoskeletal and adaptor proteins of human podocytes by interleukin-13.
- Published in:
- Inflammation Research, 2017, v. 66, n. 9, p. 793, doi. 10.1007/s00011-017-1058-y
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- Publication type:
- Article
Angiotensin II induces endoplasmic reticulum stress in podocyte, which would be further augmented by PI3-kinase inhibition.
- Published in:
- Clinical Hypertension, 2015, v. 21, n. 1, p. 1, doi. 10.1186/s40885-015-0018-5
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- Publication type:
- Article