Found: 12
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Innate and Autoimmunity in the Pathogenesis of Inherited Retinal Dystrophy.
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- Cells (2073-4409), 2020, v. 9, n. 3, p. 630, doi. 10.3390/cells9030630
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- Article
Cover Image.
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- FASEB Journal, 2024, v. 38, n. 5, p. 1, doi. 10.1096/fsb2.23571
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- Article
NUDC is critical for rod photoreceptor function, maintenance, and survival.
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- FASEB Journal, 2024, v. 38, n. 5, p. 1, doi. 10.1096/fj.202301641RR
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- Article
NudC regulates photoreceptor disk morphogenesis and rhodopsin localization.
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- FASEB Journal, 2019, v. 33, n. 8, p. 8799, doi. 10.1096/fj.201801740RR
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- Article
Mks6 mutations reveal tissue- and cell type-specific roles for the cilia transition zone.
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- FASEB Journal, 2019, v. 33, n. 1, p. 1440, doi. 10.1096/fj.201801149R
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- Article
Remodeling of the Lamina Cribrosa: Mechanisms and Potential Therapeutic Approaches for Glaucoma.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 15, p. 8068, doi. 10.3390/ijms23158068
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- Article
Autosomal dominant retinitis pigmentosa rhodopsin mutant Q344X drives specific alterations in chromatin complex gene transcription.
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- Molecular Vision, 2018, v. 24, p. 153
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- Article
Nucleotide Bound to rab11a Controls Localization in Rod Cells But Not Interaction with Rhodopsin.
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- Journal of Neuroscience, 2014, v. 34, n. 45, p. 14854, doi. 10.1523/JNEUROSCI.1943-14.2014
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- Article
Abrupt Onset of Mutations in a Developmentally Regulated Gene during Terminal Differentiation of Post-Mitotic Photoreceptor Neurons in Mice.
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- PLoS ONE, 2014, v. 9, n. 9, p. 1, doi. 10.1371/journal.pone.0108135
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- Article
Proinflammatory Pathways Are Activated in the Human Q344X Rhodopsin Knock-In Mouse Model of Retinitis Pigmentosa.
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- Biomolecules (2218-273X), 2021, v. 11, n. 8, p. 1163, doi. 10.3390/biom11081163
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- Article
Ciliary tip actin dynamics regulate photoreceptor outer segment integrity.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-48639-w
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- Article
Novel Hypomorphic Alleles of the Mouse Tyrosinase Gene Induced by CRISPR-Cas9 Nucleases Cause Non-Albino Pigmentation Phenotypes.
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- PLoS ONE, 2016, v. 11, n. 5, p. 1, doi. 10.1371/journal.pone.0155812
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- Article