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Disruption of Bioenergetics in the Intestine of Wistar Rats Caused by Hydrogen Sulfide and Thiosulfate: A Potential Mechanism of Chronic Hemorrhagic Diarrhea in Ethylmalonic Encephalopathy.
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- Cell Biochemistry & Biophysics, 2023, v. 81, n. 4, p. 683, doi. 10.1007/s12013-023-01161-0
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- Article
Myelin Disruption, Neuroinflammation, and Oxidative Stress Induced by Sulfite in the Striatum of Rats Are Mitigated by the pan-PPAR agonist Bezafibrate.
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- Cells (2073-4409), 2023, v. 12, n. 12, p. 1557, doi. 10.3390/cells12121557
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Long-chain 3-hydroxy fatty acids accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial trifunctional protein deficiencies uncouple oxidative phosphorylation in heart mitochondria.
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- Journal of Bioenergetics & Biomembranes, 2013, v. 45, n. 1/2, p. 47, doi. 10.1007/s10863-012-9481-9
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- Article
Experimental evidence that pristanic acid disrupts mitochondrial homeostasis in brain of young rats.
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- Journal of Neuroscience Research, 2012, v. 90, n. 3, p. 597, doi. 10.1002/jnr.22802
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Nuclear Factor Erythroid-Related Factor 2 Signaling in the Neuropathophysiology of Inherited Metabolic Disorders.
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- Frontiers in Cellular Neuroscience, 2021, v. 15, p. 1, doi. 10.3389/fncel.2021.785057
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Chronic postnatal ornithine administration to rats provokes learning deficit in the open field task.
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- Metabolic Brain Disease, 2012, v. 27, n. 4, p. 479, doi. 10.1007/s11011-012-9322-x
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Physical Exercise During Pregnancy Prevents Cognitive Impairment Induced by Amyloid-β in Adult Offspring Rats.
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- Molecular Neurobiology, 2019, v. 56, n. 3, p. 2022, doi. 10.1007/s12035-018-1210-x
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- Article
Bezafibrate Prevents Glycine-Induced Increase of Antioxidant Enzyme Activities in Rat Striatum.
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- Molecular Neurobiology, 2019, v. 56, n. 1, p. 29, doi. 10.1007/s12035-018-1074-0
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Maternal Hypermethioninemia Affects Neurons Number, Neurotrophins Levels, Energy Metabolism, and Na<sup>+</sup>,K<sup>+</sup>-ATPase Expression/Content in Brain of Rat Offspring.
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- Molecular Neurobiology, 2018, v. 55, n. 2, p. 980, doi. 10.1007/s12035-017-0383-z
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Glycine Administration Alters MAPK Signaling Pathways and Causes Neuronal Damage in Rat Brain: Putative Mechanisms Involved in the Neurological Dysfunction in Nonketotic Hyperglycinemia.
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- Molecular Neurobiology, 2018, v. 55, n. 1, p. 741, doi. 10.1007/s12035-016-0319-z
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- Article
Methylphenidate Decreases ATP Levels and Impairs Glutamate Uptake and Na,K-ATPase Activity in Juvenile Rat Hippocampus.
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- Molecular Neurobiology, 2017, v. 54, n. 10, p. 7796, doi. 10.1007/s12035-016-0289-1
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- Article
Intracerebral Glycine Administration Impairs Energy and Redox Homeostasis and Induces Glial Reactivity in Cerebral Cortex of Newborn Rats.
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- Molecular Neurobiology, 2016, v. 53, n. 9, p. 5864, doi. 10.1007/s12035-015-9493-7
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Evidence that glycine induces lipid peroxidation and decreases glutathione concentrations in rat cerebellum.
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- Molecular & Cellular Biochemistry, 2014, v. 395, n. 1/2, p. 125, doi. 10.1007/s11010-014-2118-z
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- Article
Phytanic acid disturbs mitochondrial homeostasis in heart of young rats: a possible pathomechanism of cardiomyopathy in Refsum disease.
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- Molecular & Cellular Biochemistry, 2012, v. 366, n. 1/2, p. 335, doi. 10.1007/s11010-012-1311-1
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In vitro evidence that sulfite impairs glutamatergic neurotransmission and inhibits glutathione metabolism-related enzymes in rat cerebral cortex.
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- International Journal of Developmental Neuroscience, 2015, v. 42, p. 68, doi. 10.1016/j.ijdevneu.2015.03.005
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- Article
Disruption of redox homeostasis in cerebral cortex of developing rats by acylcarnitines accumulating in medium-chain acyl-CoA dehydrogenase deficiency
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- International Journal of Developmental Neuroscience, 2012, v. 30, n. 5, p. 383, doi. 10.1016/j.ijdevneu.2012.03.238
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- Article
Neurochemical Evidence that Pristanic Acid Impairs Energy Production and Inhibits Synaptic Na, K-ATPase Activity in Brain of Young Rats.
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- Neurochemical Research, 2011, v. 36, n. 6, p. 1101, doi. 10.1007/s11064-011-0453-y
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- Article
Promotion of Lipid and Protein Oxidative Damage in Rat Brain by Ethylmalonic Acid.
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- Neurochemical Research, 2010, v. 35, n. 2, p. 298, doi. 10.1007/s11064-009-0055-0
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ETHE1 and MOCS1 deficiencies: Disruption of mitochondrial bioenergetics, dynamics, redox homeostasis and endoplasmic reticulum-mitochondria crosstalk in patient fibroblasts.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-49014-2
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Antioxidant properties of mesenchymal stem cells against oxidative stress in a murine model of colitis.
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- Biotechnology Letters, 2017, v. 39, n. 4, p. 613, doi. 10.1007/s10529-016-2272-3
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- Article
Medium branched chain fatty acids improve the profile of tricarboxylic acid cycle intermediates in mitochondrial fatty acid β‐oxidation deficient cells: A comparative study.
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- Journal of Inherited Metabolic Disease, 2022, v. 45, n. 3, p. 541, doi. 10.1002/jimd.12480
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The mitochondrial‐targeted reactive species scavenger JP4‐039 prevents sulfite‐induced alterations in antioxidant defenses, energy transfer, and cell death signaling in striatum of rats.
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- Journal of Inherited Metabolic Disease, 2021, v. 44, n. 2, p. 481, doi. 10.1002/jimd.12310
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Can glutathione be a biomarker for suicide risk in women 18 months postpartum?
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- Frontiers in Psychiatry, 2023, v. 14, p. 01, doi. 10.3389/fpsyt.2023.1142608
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Pulmonary arterial hypertension induces the release of circulating extracellular vesicles with oxidative content and alters redox and mitochondrial homeostasis in the brains of rats.
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- Hypertension Research, 2021, v. 44, n. 8, p. 918, doi. 10.1038/s41440-021-00660-y
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- Article
Creatine nanoliposome reverts the HPA-induced damage in complex II–III activity of the rats' cerebral cortex.
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- Molecular Biology Reports, 2019, v. 46, n. 6, p. 5897, doi. 10.1007/s11033-019-05023-y
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- Article
Sulfite Impairs Bioenergetics and Redox Status in Neonatal Rat Brain: Insights into the Early Neuropathophysiology of Isolated Sulfite Oxidase and Molybdenum Cofactor Deficiencies.
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- Cellular & Molecular Neurobiology, 2023, v. 43, n. 6, p. 2895, doi. 10.1007/s10571-023-01328-6
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Mitochondrial Dysfunction and Redox Homeostasis Impairment as Pathomechanisms of Brain Damage in Ethylmalonic Encephalopathy: Insights from Animal and Human Studies.
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- Cellular & Molecular Neurobiology, 2022, v. 42, n. 3, p. 565, doi. 10.1007/s10571-020-00976-2
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- Article
Neurochemical Evidence that the Metabolites Accumulating in 3-Methylcrotonyl-CoA Carboxylase Deficiency Induce Oxidative Damage in Cerebral Cortex of Young Rats.
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- Cellular & Molecular Neurobiology, 2013, v. 33, n. 1, p. 137, doi. 10.1007/s10571-012-9879-2
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- Article
Lipopolysaccharide-Elicited Systemic Inflammation Induces Selective Vulnerability of Cerebral Cortex and Striatum of Developing Glutaryl-CoA Dehydrogenase Deficient (Gcdh<sup>−/−</sup>) Mice to Oxidative Stress.
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- Neurotoxicity Research, 2020, v. 38, n. 4, p. 1024, doi. 10.1007/s12640-020-00291-0
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- Article
3-Hydroxy-3-Methylglutaric Acid Impairs Redox and Energy Homeostasis, Mitochondrial Dynamics, and Endoplasmic Reticulum–Mitochondria Crosstalk in Rat Brain.
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- Neurotoxicity Research, 2020, v. 37, n. 2, p. 314, doi. 10.1007/s12640-019-00122-x
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The Role of Oxidative Stress and Bioenergetic Dysfunction in Sulfite Oxidase Deficiency: Insights from Animal Models.
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- Neurotoxicity Research, 2019, v. 35, n. 2, p. 484, doi. 10.1007/s12640-018-9986-z
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- Article
Evidence that Thiosulfate Inhibits Creatine Kinase Activity in Rat Striatum via Thiol Group Oxidation.
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- Neurotoxicity Research, 2018, v. 34, n. 3, p. 693, doi. 10.1007/s12640-018-9934-y
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- Article
Disruption of Energy Transfer and Redox Status by Sulfite in Hippocampus, Striatum, and Cerebellum of Developing Rats.
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- Neurotoxicity Research, 2017, v. 32, n. 2, p. 264, doi. 10.1007/s12640-017-9732-y
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- Article
Glycine Intracerebroventricular Administration Disrupts Mitochondrial Energy Homeostasis in Cerebral Cortex and Striatum of Young Rats.
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- Neurotoxicity Research, 2013, v. 24, n. 4, p. 502, doi. 10.1007/s12640-013-9396-1
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- Article