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Neuroprotective and cerebrovascular effects of endogenous N-Arachidonoyl-GABA and its putative Cox-2 metabolite -- GABA conjugate with Prostaglandin E2.
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- Research Results in Pharmacology, 2021, v. 7, n. 3, p. 49, doi. 10.3897/rrpharmacology.7.70974
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- Article
N‐arachidonoyl dopamine inhibits epithelial–mesenchymal transition of breast cancer cells through ERK signaling and decreasing the cellular cholesterol.
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- Journal of Biochemical & Molecular Toxicology, 2021, v. 35, n. 4, p. 1, doi. 10.1002/jbt.22693
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- Article
Arachidonoylcholine and Other Unsaturated Long-Chain Acylcholines Are Endogenous Modulators of the Acetylcholine Signaling System.
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- Biomolecules (2218-273X), 2020, v. 10, n. 2, p. 283, doi. 10.3390/biom10020283
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- Article
The Interaction of the Endocannabinoid Anandamide and Paracannabinoid Lysophosphatidylinositol during Cell Death Induction in Human Breast Cancer Cells.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 4, p. 2271, doi. 10.3390/ijms25042271
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- Article
The Mechanisms of GPR55 Receptor Functional Selectivity during Apoptosis and Proliferation Regulation in Cancer Cells.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 6, p. 5524, doi. 10.3390/ijms24065524
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- Article
A New Method for the Visualization of Living Dopaminergic Neurons and Prospects for Using It to Develop Targeted Drug Delivery to These Cells.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 7, p. 3678, doi. 10.3390/ijms23073678
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- Article
N -Acyl Dopamines Induce Apoptosis in Endometrial Stromal Cells from Patients with Endometriosis.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 19, p. 10648, doi. 10.3390/ijms221910648
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- Article
GPR55 Receptor Activation by the N -Acyl Dopamine Family Lipids Induces Apoptosis in Cancer Cells via the Nitric Oxide Synthase (nNOS) Over-Stimulation.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 2, p. 622, doi. 10.3390/ijms22020622
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- Article