Found: 7
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Skeletal muscle delimited myopathy and verapamil toxicity in SUR2 mutant mouse models of AIMS.
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- EMBO Molecular Medicine, 2023, v. 15, n. 6, p. 1, doi. 10.15252/emmm.202216883
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- Article
Ca[subv]1.4α1 Subunits Can Form Slowly Inactivating Dihydropyridine-Sensitive L-Type Ca<sup>2+</sup> Channels Lacking Ca<sup>2+</sup>-Dependent Inactivation.
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- Journal of Neuroscience, 2003, v. 23, n. 14, p. 6041, doi. 10.1523/JNEUROSCI.23-14-06041.2003
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- Article
The familial hemiplegic migraine mutation R192q reduces G-protein-mediated inhibition of p/q-type (Ca<sub>v</sub>2.1) calcium channels expressed in human embryonic kidney cells.
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- Journal of Physiology, 2003, v. 546, n. 2, p. 337, doi. 10.1113/jphysiol.2002.026716
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- Article
Pore mutation N617D in the skeletal muscle DHPR blocks Ca<sup>2+</sup> influx due to atypical high-affinity Ca<sup>2+</sup> binding.
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- eLife, 2021, p. 1, doi. 10.7554/eLife.63435
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- Article
The mechanism underlying transient weakness in myotonia congenita.
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- eLife, 2021, p. 1, doi. 10.7554/eLife.65691
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- Article
Ca<sup>2+</sup>-activated Cl<sup>−</sup> channel TMEM16A/ANO1 identified in zebrafish skeletal muscle is crucial for action potential acceleration.
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- Nature Communications, 2019, v. 10, n. 1, p. 1, doi. 10.1038/s41467-018-07918-z
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- Article
The Ca<sup>2+</sup> influx through the mammalian skeletal muscle dihydropyridine receptor is irrelevant for muscle performance.
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- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-00629-x
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- Article