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TLR2 Activation by Porphyromonas gingivalis Requires Both PPAD Activity and Fimbriae.
- Published in:
- Frontiers in Immunology, 2024, p. 1, doi. 10.3389/fimmu.2022.823685
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- Publication type:
- Article
Identification of a new genetic variant (G231N, E232T, N235D) of peptidylarginine deiminase from P. gingivalis in advanced periodontitis.
- Published in:
- Frontiers in Immunology, 2024, p. 01, doi. 10.3389/fimmu.2024.1355357
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- Publication type:
- Article
Gingival fibroblast activation by Porphyromonas gingivalis is driven by TLR2 and is independent of the LPS‐TLR4 axis.
- Published in:
- European Journal of Immunology, 2024, v. 54, n. 3, p. 1, doi. 10.1002/eji.202350776
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- Article
Accessory fimbrial subunits and PPAD are necessary for TLR2 activation by Porphyromonas gingivalis.
- Published in:
- Molecular Oral Microbiology, 2023, v. 38, n. 4, p. 334, doi. 10.1111/omi.12427
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- Publication type:
- Article
Mapping of DNA methylation-sensitive cellular processes in gingival and periodontal ligament fibroblasts in the context of periodontal tissue homeostasis.
- Published in:
- Frontiers in Immunology, 2023, v. 13, p. 1, doi. 10.3389/fimmu.2023.1078031
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- Publication type:
- Article
Matrix Metalloproteinase-12 Supports Pulmonary B Cell Follicle Formation and Local Antibody Responses During Asthma.
- Published in:
- American Journal of Respiratory & Critical Care Medicine, 2022, v. 206, n. 11, p. 1424, doi. 10.1164/rccm.202109-2082le
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- Publication type:
- Article
TLR2 Activation by Porphyromonas gingivalis Requires Both PPAD Activity and Fimbriae.
- Published in:
- Frontiers in Immunology, 2022, v. 13, p. 1, doi. 10.3389/fimmu.2022.823685
- By:
- Publication type:
- Article
Clinics and genetic background of hereditary gingival fibromatosis.
- Published in:
- 2021
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- Publication type:
- journal article
hTERT-immortalized gingival fibroblasts respond to cytokines but fail to mimic primary cell responses to Porphyromonas gingivalis.
- Published in:
- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-90037-5
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- Publication type:
- Article
Epigenetic regulation of inflammation in periodontitis: cellular mechanisms and therapeutic potential.
- Published in:
- Clinical Epigenetics, 2020, v. 12, n. 1, p. N.PAG, doi. 10.1186/s13148-020-00982-7
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- Publication type:
- Article
Application of the In Vitro HoxB8 Model System to Characterize the Contributions of Neutrophil–LPS Interaction to Periodontal Disease.
- Published in:
- Pathogens, 2020, v. 9, n. 7, p. 530, doi. 10.3390/pathogens9070530
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- Publication type:
- Article
TIMP‐1 association with collagen type I overproduction in hereditary gingival fibromatosis.
- Published in:
- Oral Diseases, 2018, v. 24, n. 8, p. 1581, doi. 10.1111/odi.12938
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- Publication type:
- Article
An extracellular matrix fragment drives epithelial remodeling and airway hyperresponsiveness.
- Published in:
- Science Translational Medicine, 2018, v. 10, n. 455, p. 1, doi. 10.1126/scitranslmed.aaq0693
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- Publication type:
- Article
Control of cytokine mRNA degradation by the histone deacetylase inhibitor ITF2357 in rheumatoid arthritis fibroblast-like synoviocytes: beyond transcriptional regulation.
- Published in:
- Arthritis Research & Therapy, 2018, v. 20, n. 1, p. N.PAG, doi. 10.1186/s13075-018-1638-4
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- Publication type:
- Article
Axl and MerTK receptor tyrosine kinases maintain human macrophage efferocytic capacity in the presence of viral triggers.
- Published in:
- European Journal of Immunology, 2018, v. 48, n. 5, p. 855, doi. 10.1002/eji.201747283
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- Publication type:
- Article
Endoplasmic reticulum stress cooperates with Toll-like receptor ligation in driving activation of rheumatoid arthritis fibroblast-like synoviocytes.
- Published in:
- Arthritis Research & Therapy, 2017, v. 19, p. 1, doi. 10.1186/s13075-017-1386-x
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- Publication type:
- Article