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Molecular Aspects Involved in the Mechanisms of Bothrops jararaca Venom-Induced Hyperalgesia: Participation of NK1 Receptor and Glial Cells.
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- Toxins, 2024, v. 16, n. 4, p. 187, doi. 10.3390/toxins16040187
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Oligopeptidases B from Trypanossoma cruzi and Trypanossoma brucei Inhibit Inflammatory Pain in Mice by Targeting Serotoninergic Receptors.
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- Inflammation, 2013, v. 36, n. 3, p. 705, doi. 10.1007/s10753-013-9595-1
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- Article
Neutrophilic Cell-Free Exudate Induces Antinociception Mediate by the Protein S100A9.
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- Mediators of Inflammation, 2006, v. 2006, n. 4, p. 1, doi. 10.1155/MI/2006/36765
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Neutrophilic Cell-Free Exudate Induces Antinociception Mediate by the Protein S100A9.
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- Mediators of Inflammation, 2006, v. 2006, p. 1, doi. 10.1155/MI/2006/36765
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- Article
Neutrophils and the calcium-binding protein MRP-14 mediate carrageenan-induced antinociception in mice.
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- Mediators of Inflammation, 2002, v. 11, n. 4, p. 203, doi. 10.1080/0962935029000050
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- Article
Analgesic properties of S100A9 C-terminal domain: a mechanism dependent on calcium channel inhibition.
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- Fundamental & Clinical Pharmacology, 2009, v. 23, n. 4, p. 427, doi. 10.1111/j.1472-8206.2009.00686.x
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Inhibition of Macrophage Functions by the C-Terminus of Murine S100A9 Is Dependent on B-1 Cells.
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- Mediators of Inflammation, 2014, v. 2014, p. 1, doi. 10.1155/2014/836491
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