BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis.
- Published in:
- Cell Death Discovery, 2018, v. 4, n. 1, p. N.PAG, doi. 10.1038/s41420-018-0054-5
- By:
- Publication type:
- Article
Works by Gerasimenko, Julia V.
Results: 17
1
2
NAADP mobilizes Ca[SUP2+] from a thapsigargin-sensitive store in the nuclear envelope by activating ryanodine receptors.
- Published in:
- Journal of Cell Biology, 2003, v. 163, n. 2, p. 271, doi. 10.1083/jcb.200306134
- By:
- Publication type:
- Article
3
Activation of pancreatic stellate cells attenuates intracellular Ca<sup>2+</sup> signals due to downregulation of TRPA1 and protects against cell death induced by alcohol metabolites.
- Published in:
- Cell Death & Disease, 2022, v. 13, n. 8, p. 1, doi. 10.1038/s41419-022-05186-w
- By:
- Publication type:
- Article
4
Role of intracellular acid Ca<sup>2+</sup> stores in pathological pancreatic protease activation.
- Published in:
- 2012
- By:
- Publication type:
- Editorial
5
Two different but converging messenger pathways to intracellular Ca<sup>2+</sup> release: the roles of nicotinic acid adenine dinucleotide phosphate, cyclic ADP-ribose and inositol trisphosphate.
- Published in:
- EMBO Journal, 2000, v. 19, n. 11, p. 2549, doi. 10.1093/emboj/19.11.2549
- By:
- Publication type:
- Article
6
Active mitochondria surrounding the pancreatic acinar granule region prevent spreading of inositol trisphosphate-evoked local cytosolic Ca<sup>2+</sup> signals.
- Published in:
- EMBO Journal, 1999, v. 18, n. 18, p. 4999, doi. 10.1093/emboj/18.18.4999
- By:
- Publication type:
- Article
7
ABT-199 (Venetoclax), a BH3-mimetic Bcl-2 inhibitor, does not cause Ca<sup>2+</sup> -signalling dysregulation or toxicity in pancreatic acinar cells.
- Published in:
- 2019
- By:
- Publication type:
- journal article
8
The role of CFTR in diabetes‐induced pancreatic ductal dysfunction.
- Published in:
- Journal of Physiology, 2024, v. 602, n. 6, p. 993, doi. 10.1113/JP286338
- By:
- Publication type:
- Article
9
CRAC channel inhibitors in pancreatic pathologies.
- Published in:
- Journal of Physiology, 2022, v. 600, n. 7, p. 1597, doi. 10.1113/JP282826
- By:
- Publication type:
- Article
10
Calcium signalling in the acinar environment of the exocrine pancreas: physiology and pathophysiology.
- Published in:
- Journal of Physiology, 2018, v. 596, n. 14, p. 2663, doi. 10.1113/JP275395
- By:
- Publication type:
- Article
11
Bile acids induce necrosis in pancreatic stellate cells dependent on calcium entry and sodium-driven bile uptake.
- Published in:
- Journal of Physiology, 2016, v. 594, n. 21, p. 6147, doi. 10.1113/JP272774
- By:
- Publication type:
- Article
12
Ca<sup>2+</sup> signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca<sup>2+</sup> channel blockade.
- Published in:
- Journal of Physiology, 2016, v. 594, n. 2, p. 281, doi. 10.1113/JP271468
- By:
- Publication type:
- Article
13
The role of Ca<sup>2+</sup> in the pathophysiology of pancreatitis.
- Published in:
- Journal of Physiology, 2014, v. 592, n. 2, p. 269, doi. 10.1113/jphysiol.2013.261784
- By:
- Publication type:
- Article
14
Ca<sup>2+</sup> Signaling and ATP Production in Pancreatic Cancer.
- Published in:
- Function, 2024, v. 5, n. 1, p. 1, doi. 10.1093/function/zqad067
- By:
- Publication type:
- Article
15
SARS-CoV-2 S Protein Subunit 1 Elicits Ca2+ Influx – Dependent Ca2+ Signals in Pancreatic Stellate Cells and Macrophages In Situ.
- Published in:
- Function, 2022, v. 3, n. 2, p. 1, doi. 10.1093/function/zqac002
- By:
- Publication type:
- Article
16
Endocytic uptake of SARS-CoV-2: the critical roles of pH, Ca<sup>2+</sup>, and NAADP.
- Published in:
- Function, 2020, v. 1, n. 1, p. 1, doi. 10.1093/function/zqaa003
- By:
- Publication type:
- Article
17
Galactose protects against cell damage in mouse models of acute pancreatitis.
- Published in:
- 2018
- By:
- Publication type:
- journal article