Found: 21
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Intra-islet proliferation of cytotoxic T lymphocytes contributes to insulitis progression.
- Published in:
- European Journal of Immunology, 2012, v. 42, n. 7, p. 1717, doi. 10.1002/eji.201242435
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- Publication type:
- Article
Deficiency of the innate immune adaptor STING promotes autoreactive T cell expansion in NOD mice.
- Published in:
- Diabetologia, 2021, v. 64, n. 4, p. 878, doi. 10.1007/s00125-020-05378-z
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- Publication type:
- Article
Autoreactive T cells induce necrosis and not BCL-2-regulated or death receptor-mediated apoptosis or RIPK3-dependent necroptosis of transplanted islets in a mouse model of type 1 diabetes.
- Published in:
- Diabetologia, 2015, v. 58, n. 1, p. 140, doi. 10.1007/s00125-014-3407-5
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- Publication type:
- Article
Activation of the NLRP3 Inflammasome Complex is Not Required for Stress-Induced Death of Pancreatic Islets.
- Published in:
- PLoS ONE, 2014, v. 9, n. 11, p. 1, doi. 10.1371/journal.pone.0113128
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- Article
ABCA12 regulates insulin secretion from β‐cells.
- Published in:
- EMBO Reports, 2020, v. 21, n. 3, p. 1, doi. 10.15252/embr.201948692
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- Publication type:
- Article
IL-21 regulates SOCS1 expression in autoreactive CD8<sup>+</sup> T cells but is not required for acquisition of CTL activity in the islets of non-obese diabetic mice.
- Published in:
- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-51636-5
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- Publication type:
- Article
The JAK1 Selective Inhibitor ABT 317 Blocks Signaling Through Interferon-γ and Common γ Chain Cytokine Receptors to Reverse Autoimmune Diabetes in NOD Mice.
- Published in:
- Frontiers in Immunology, 2020, v. 11, p. N.PAG, doi. 10.3389/fimmu.2020.588543
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- Publication type:
- Article
IL-17F induces inflammation, dysfunction and cell death in mouse islets.
- Published in:
- Scientific Reports, 2020, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41598-020-69805-2
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- Publication type:
- Article
Soluble FAS ligand is not required for pancreatic islet inflammation or beta-cell destruction in non-obese diabetic mice.
- Published in:
- Cell Death Discovery, 2019, v. 5, n. 1, p. N.PAG, doi. 10.1038/s41420-019-0217-z
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- Publication type:
- Article
Diabetes induced by checkpoint inhibition in nonobese diabetic mice can be prevented or reversed by a JAK1/JAK2 inhibitor.
- Published in:
- Clinical & Translational Immunology, 2022, v. 11, n. 11, p. 1, doi. 10.1002/cti2.1425
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- Publication type:
- Article
The pro-apoptotic BH3-only protein Bid is dispensable for development of insulitis and diabetes in the non-obese diabetic mouse.
- Published in:
- Apoptosis, 2011, v. 16, n. 8, p. 822, doi. 10.1007/s10495-011-0615-z
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- Publication type:
- Article
Perforin facilitates beta cell killing and regulates autoreactive CD8<sup>+</sup> T-cell responses to antigen in mouse models of type 1 diabetes.
- Published in:
- Immunology & Cell Biology, 2016, v. 94, n. 4, p. 334, doi. 10.1038/icb.2015.89
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- Publication type:
- Article
Granzyme A Deficiency Breaks Immune Tolerance and Promotes Autoimmune Diabetes Through a Type I Interferon-Dependent Pathway.
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- 2017
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- Publication type:
- journal article
Repurposed JAK1/JAK2 Inhibitor Reverses Established Autoimmune Insulitis in NOD Mice.
- Published in:
- 2017
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- Publication type:
- journal article
Deficiency in Type I Interferon Signaling Prevents the Early Interferon--Induced Gene Signature in Pancreatic Islets but Not Type 1 Diabetes in NOD Mice.
- Published in:
- Diabetes, 2014, v. 63, n. 3, p. 1032, doi. 10.2337/db13-1210
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- Publication type:
- Article
The protective effects of CD39 overexpression in multiple low-dose streptozotocin-induced diabetes in mice.
- Published in:
- 2013
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- Publication type:
- journal article
The Protective Effects of CD39 Overexpression in Multiple Low-Dose Streptozotocin--Induced Diabetes in Mice.
- Published in:
- Diabetes, 2013, v. 62, n. 6, p. 2026, doi. 10.2337/db12-0625
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- Publication type:
- Article
Complete Diabetes Protection Despite Delayed Thymic Tolerance in NOD8.3 TCR Transgenic Mice Due to Antigen-Induced Extrathymic Deletion of T Cells.
- Published in:
- Diabetes, 2012, v. 61, n. 2, p. 425, doi. 10.2337/db11-0948
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- Publication type:
- Article
Macrophage Deletion of SOCS1 Increases Sensitivity to LPS and Palmitic Acid and Results in Systemic Inflammation and Hepatic Insulin Resistance.
- Published in:
- Diabetes, 2011, v. 60, n. 8, p. 2023, doi. 10.2337/db11-0259
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- Publication type:
- Article
Islet beta-cells deficient in Bcl-xL develop but are abnormally sensitive to apoptotic stimuli.
- Published in:
- 2009
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- Publication type:
- journal article
Islet β-Cells Deficient in Bcl-xL Develop but Are Abnormally Sensitive to Apoptotic Stimuli.
- Published in:
- Diabetes, 2009, v. 58, n. 10, p. 2316, doi. 10.2337/db08-1602
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- Publication type:
- Article