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PQBP5/NOL10 maintains and anchors the nucleolus under physiological and osmotic stress conditions.
- Published in:
- Nature Communications, 2023, v. 14, n. 1, p. 1, doi. 10.1038/s41467-022-35602-w
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- Article
CD38 is critical for social behaviour by regulating oxytocin secretion.
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- Nature, 2007, v. 446, n. 7131, p. 41, doi. 10.1038/nature05526
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- Article
Comprehensive phosphoproteome analysis unravels the core signaling network that initiates the earliest synapse pathology in preclinical Alzheimer’s disease brain.
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- Human Molecular Genetics, 2015, v. 24, n. 2, p. 540, doi. 10.1093/hmg/ddu475
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- Article
Systems biology analysis of Drosophila in vivo screen data elucidates core networks for DNA damage repair in SCA1.
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- Human Molecular Genetics, 2014, v. 23, n. 5, p. 1345, doi. 10.1093/hmg/ddt524
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- Article
Targeting Tyro3 ameliorates a model of PGRNmutant FTLD-TDP via tau-mediated synaptic pathology.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-02821-z
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- Article
Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1.
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- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-01790-z
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- Article
A functional deficiency of TERA/VCP/p97 contributes to impaired DNA repair in multiple polyglutamine diseases.
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- Nature Communications, 2013, v. 4, n. 5, p. 1816, doi. 10.1038/ncomms2828
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- Article
Necrosis Links Neurodegeneration and Neuroinflammation in Neurodegenerative Disease.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 7, p. 3636, doi. 10.3390/ijms25073636
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- Article
IL-6 Receptor Is a Possible Target against Growth of Metastasized Lung Tumor Cells in the Brain.
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- International Journal of Molecular Sciences, 2013, v. 14, n. 1, p. 515, doi. 10.3390/ijms14010515
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- Article
Interaction between lung cancer cells and astrocytes via specific inflammatory cytokines in the microenvironment of brain metastasis.
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- Clinical & Experimental Metastasis, 2011, v. 28, n. 1, p. 13, doi. 10.1007/s10585-010-9354-8
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- Article
HMGB1 facilitates repair of mitochondrial DNA damage and extends the lifespan of mutant ataxin-1 knock-in mice.
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- EMBO Molecular Medicine, 2015, v. 7, n. 1, p. 78, doi. 10.15252/emmm.201404392
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- Article
Drebrin-like (Dbnl) Controls Neuronal Migration via Regulating N-Cadherin Expression in the Developing Cerebral Cortex.
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- Journal of Neuroscience, 2019, v. 39, n. 4, p. 678, doi. 10.1523/JNEUROSCI.1634-18.2018
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- Article
Sox2 Transcriptionally Regulates Pqbp1, an Intellectual Disability-Microcephaly Causative Gene, in Neural Stem Progenitor Cells.
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- PLoS ONE, 2013, v. 8, n. 7, p. 1, doi. 10.1371/journal.pone.0068627
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- Article
Hydrogen in Drinking Water Reduces Dopaminergic Neuronal Loss in the 1-methyl-4-phenyl-1,2,3,6- tetrahydropyridine Mouse Model of Parkinson's Disease.
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- PLoS ONE, 2009, v. 4, n. 9, p. 1, doi. 10.1371/journal.pone.0007247
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- Article
Effects of 3,3',5-triiodothyronine on microglial functions.
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- Glia, 2015, v. 63, n. 5, p. 906, doi. 10.1002/glia.22792
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- Article
Expression, subunit composition, and function of AMPA-type glutamate receptors are changed in activated microglia; possible contribution of GluA2 (GluR-B)-deficiency under pathological conditions.
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- Glia, 2013, v. 61, n. 6, p. 881, doi. 10.1002/glia.22481
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- Article
Identification of hepta-histidine as a candidate drug for Huntington's disease by in silico-in vitro- in vivo-integrated screens of chemical libraries.
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- Scientific Reports, 2016, p. 33861, doi. 10.1038/srep33861
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- Article
HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer's disease.
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- Scientific Reports, 2016, p. 31895, doi. 10.1038/srep31895
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- Article
Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation.
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- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-26851-2
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- Article
HMGB1 signaling phosphorylates Ku70 and impairs DNA damage repair in Alzheimer's disease pathology.
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- Communications Biology, 2021, v. 4, n. 1, p. 1, doi. 10.1038/s42003-021-02671-4
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- Article
A novel form of necrosis, TRIAD, occurs in human Huntington's disease.
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- Acta Neuropathologica Communications, 2017, v. 5, p. 1, doi. 10.1186/s40478-017-0420-1
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- Article
YAP-dependent necrosis occurs in early stages of Alzheimer's disease and regulates mouse model pathology.
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- Nature Communications, 2020, v. 11, n. 1, p. 1, doi. 10.1038/s41467-020-14353-6
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- Article
Role of the Drosophila YATA protein in the proper subcellular localization of COPI revealed by in vivo analysis.
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- Genes & Genetic Systems, 2020, v. 95, n. 6, p. 303, doi. 10.1266/ggs.20-00027
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- Article
Targeting TEAD/YAP-transcription-dependent necrosis, TRIAD, ameliorates Huntington’s disease pathology.
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- Human Molecular Genetics, 2016, v. 25, n. 21, p. 4749, doi. 10.1093/hmg/ddw303
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- Article
Mild Maternal Hypothyroxinemia During Pregnancy Induces Persistent DNA Hypermethylation in the Hippocampal Brain-Derived Neurotrophic Factor Gene in Mouse Offspring.
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- Thyroid, 2018, v. 28, n. 3, p. 395, doi. 10.1089/thy.2017.0331
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- Article
Therapeutic Approach to Neurodegenerative Diseases by Medical Gases: Focusing on Redox Signaling and Related Antioxidant Enzymes.
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- Oxidative Medicine & Cellular Longevity, 2012, v. 2012, p. 1, doi. 10.1155/2012/324256
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- Article
Dynamic molecular network analysis of iPSC-Purkinje cells differentiation delineates roles of ISG15 in SCA1 at the earliest stage.
- Published in:
- Communications Biology, 2024, v. 7, n. 1, p. 1, doi. 10.1038/s42003-024-06066-z
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- Article