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Acetylation of KLF5 maintains EMT and tumorigenicity to cause chemoresistant bone metastasis in prostate cancer.
- Published in:
- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-21976-w
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- Article
Deciphering intercellular signaling complexes by interaction-guided chemical proteomics.
- Published in:
- Nature Communications, 2023, v. 14, n. 1, p. 1, doi. 10.1038/s41467-023-39881-9
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- Article
Saturated fatty acids increase LPI to reduce FUNDC1 dimerization and stability and mitochondrial function.
- Published in:
- EMBO Reports, 2023, v. 24, n. 4, p. 1, doi. 10.15252/embr.202254731
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- Article
KLF5 Is Crucial for Androgen-AR Signaling to Transactivate Genes and Promote Cell Proliferation in Prostate Cancer Cells.
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- Cancers, 2020, v. 12, n. 3, p. 748, doi. 10.3390/cancers12030748
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- Article
CINP is a novel cofactor of KLF5 required for its role in the promotion of cell proliferation, survival and tumor growth.
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- International Journal of Cancer, 2019, v. 144, n. 3, p. 582, doi. 10.1002/ijc.31908
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- Article
Kindlin-2 enhances c-Myc translation through association with DDX3X to promote pancreatic ductal adenocarcinoma progression.
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- Theranostics, 2023, v. 13, n. 13, p. 4333, doi. 10.7150/thno.85421
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- Article
ZFHX3 is indispensable for ERβ to inhibit cell proliferation via MYC downregulation in prostate cancer cells.
- Published in:
- Oncogenesis, 2019, v. 8, n. 4, p. N.PAG, doi. 10.1038/s41389-019-0138-y
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- Article