Found: 11
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Signatures of disrupted synaptic maintenance in the entorhinal cortex of both pathology‐free APOE4 carriers and aged APOE4 mice: Molecular and cell biology/synaptic disruption.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2020, v. 16, n. 11, p. 1, doi. 10.1002/alz.046192
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- Article
P3‐175: APOE4 INDUCES REGION‐SPECIFIC DYSFUNCTION IN THE ENTORHINAL CORTEX OF AGED APOE4 MICE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P997, doi. 10.1016/j.jalz.2019.06.3204
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- Article
O2‐01‐04: CELL TYPE–SPECIFIC TAU HOMEOSTASIS SIGNATURES ASSOCIATED WITH SELECTIVE VULNERABILITY OF EXCITATORY NEURONS TO TAU PATHOLOGY.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P609, doi. 10.1016/j.jalz.2018.06.2642
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- Article
The Endosomal-Lysosomal Pathway Is Dysregulated by APOE4 Expression in Vivo.
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- Frontiers in Neuroscience, 2017, p. 1, doi. 10.3389/fnins.2017.00702
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- Article
Anesthesia-Induced Hyperphosphorylation Detaches 3-Repeat Tau from Microtubules without Affecting Their Stability In Vivo.
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- Journal of Neuroscience, 2008, v. 28, n. 48, p. 12798, doi. 10.1523/JNEUROSCI.4101-08.2008
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- Article
Interplay between Cyclin-Dependent Kinase 5 and Glycogen Synthase Kinase 3β Mediated by Neuregulin Signaling Leads to Differential Effects on Tau Phosphorylation and Amyloid Precursor Protein Processing.
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- Journal of Neuroscience, 2008, v. 28, n. 10, p. 2624, doi. 10.1523/JNEUROSCI.5245-07.2008
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- Article
APOE4 is Associated with Differential Regional Vulnerability to Bioenergetic Deficits in Aged APOE Mice.
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- Scientific Reports, 2020, v. 10, n. 1, p. 1, doi. 10.1038/s41598-020-61142-8
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- Article
3D Visualization of the Temporal and Spatial Spread of Tau Pathology Reveals Extensive Sites of Tau Accumulation Associated with Neuronal Loss and Recognition Memory Deficit in Aged Tau Transgenic Mice.
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- PLoS ONE, 2016, v. 11, n. 7, p. 1, doi. 10.1371/journal.pone.0159463
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- Article
Neuronal hyperactivity due to loss of inhibitory tone in APOE4 mice lacking Alzheimer’s diseaselike pathology.
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- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-01444-0
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- Article
PAC1 receptor–mediated clearance of tau in postsynaptic compartments attenuates tau pathology in mouse brain.
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- Science Translational Medicine, 2021, v. 13, n. 595, p. 1, doi. 10.1126/scitranslmed.aba7394
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- Article
5-HT4 receptor agonists treatment reduces tau pathology and behavioral de?cit in the PS19 mouse model of tauopathy.
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- Frontiers in Cellular Neuroscience, 2024, p. 01, doi. 10.3389/fncel.2024.1338502
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- Article