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Renal hypoxia due to increased oxygen metabolism is an independent pathway to nephropathy (890.6).
- Published in:
- FASEB Journal, 2014, v. 28, p. N.PAG, doi. 10.1096/fasebj.28.1_supplement.890.6
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- Article
Decreased kidney oxygenation due to mitochondrial uncoupling: a mechanism to injury after renal transplantation (890.7).
- Published in:
- FASEB Journal, 2014, v. 28, p. N.PAG, doi. 10.1096/fasebj.28.1_supplement.890.7
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- Article
Thyroid hormone increases oxygen metabolism causing intrarenal tissue hypoxia; a pathway to kidney disease.
- Published in:
- PLoS ONE, 2022, v. 17, n. 3, p. 1, doi. 10.1371/journal.pone.0264524
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- Article
Adipocyte-Mineralocorticoid Receptor Alters Mitochondrial Quality Control Leading to Mitochondrial Dysfunction and Senescence of Visceral Adipose Tissue.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 6, p. 2881, doi. 10.3390/ijms22062881
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- Article
Hyaluronan Production by Renomedullary Interstitial Cells: Influence of Endothelin, Angiotensin II and Vasopressin.
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- International Journal of Molecular Sciences, 2017, v. 18, n. 12, p. 2701, doi. 10.3390/ijms18122701
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- Article
Mitochondrial Reactive Oxygen Species and Kidney Hypoxia in the Development of Diabetic Nephropathy.
- Published in:
- Frontiers in Physiology, 2017, p. 1, doi. 10.3389/fphys.2017.00211
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- Article
Acute Knockdown of Uncoupling Protein-2 Increases Uncoupling via the Adenine Nucleotide Transporter and Decreases Oxidative Stress in Diabetic Kidneys.
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- PLoS ONE, 2012, v. 7, n. 7, p. 1, doi. 10.1371/journal.pone.0039635
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- Article
Determinants of renal oxygen metabolism during low Na<sup>+</sup> diet: effect of angiotensin II AT<sub>1</sub> and aldosterone receptor blockade.
- Published in:
- Journal of Physiology, 2020, v. 598, n. 23, p. 5573, doi. 10.1113/JP280481
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- Article