Found: 11
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ECM and epithelial stem cells: the scaffold of destiny.
- Published in:
- Frontiers in Cell & Developmental Biology, 2024, p. 1, doi. 10.3389/fcell.2024.1359585
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- Publication type:
- Article
Fibroblast growth factor-2 bound to specific dermal fibroblast-derived extracellular vesicles is protected from degradation.
- Published in:
- Scientific Reports, 2022, v. 12, n. 1, p. 1, doi. 10.1038/s41598-022-26217-8
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- Publication type:
- Article
Cell metabolism regulates integrin mechanosensing via an SLC3A2-dependent sphingolipid biosynthesis pathway.
- Published in:
- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-07268-w
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- Publication type:
- Article
Adipocytes secrete leukotrienes: contribution to obesity-associated inflammation and insulin resistance in mice.
- Published in:
- 2012
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- Publication type:
- journal article
Adipocytes Secrete Leukotrienes.
- Published in:
- Diabetes, 2012, v. 61, n. 9, p. 2311, doi. 10.2337/db11-1455
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- Publication type:
- Article
Off the beaten paths: alternative and crosstalk regulation of Rho GTPases.
- Published in:
- FASEB Journal, 2012, v. 26, n. 2, p. 469, doi. 10.1096/fj.11-192252
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- Publication type:
- Article
CD98hc facilitates B cell proliferation and adaptive humoral immunity.
- Published in:
- Nature Immunology, 2009, v. 10, n. 4, p. 412, doi. 10.1038/ni.1712
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- Publication type:
- Article
Blockade of alpha4 integrin signaling ameliorates the metabolic consequences of high-fat diet-induced obesity.
- Published in:
- 2008
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- Publication type:
- journal article
Blockade of α4 Integrin Signaling Ameliorates the Metabolic Consequences of High-Fat Diet--Induced Obesity.
- Published in:
- Diabetes, 2008, v. 57, n. 7, p. 1842, doi. 10.2337/db07-1751
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- Publication type:
- Article
CD98hc (SLC3A2) participates in fibronectin matrix assembly by mediating integrin signaling.
- Published in:
- Journal of Cell Biology, 2007, v. 178, n. 4, p. 701
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- Publication type:
- Article
Blocking the α4 integrin-paxillin interaction selectively impairs mononuclear leukocyte recruitment to an inflammatory site.
- Published in:
- Journal of Clinical Investigation, 2006, v. 116, n. 3, p. 715, doi. 10.1172/JCI26091
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- Publication type:
- Article