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FAM83B is a novel biomarker for diagnosis and prognosis of lung squamous cell carcinoma.
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- International Journal of Oncology, 2015, v. 46, n. 3, p. 999, doi. 10.3892/ijo.2015.2817
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Effects of X-ray exposure in 3 strains of mice: acute, late and repeated low dose.
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- Experimental Animals, 2022, v. 71, p. 111
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- Article
Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice.
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- Journal of Cell Biology, 2005, v. 169, n. 3, p. 425, doi. 10.1083/jcb.200412022
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- Article
CLC-3 deficiency leads to phenotypes similar to human neuronal ceroid lipofuscinosis.
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- Genes to Cells, 2002, v. 7, n. 6, p. 597, doi. 10.1046/j.1365-2443.2002.00539.x
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- Article
Accumulation of mitochondrial ATP synthase subunit c in muscle in a patient with neuronal ceroid lipofuscinosis (late infantile form).
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- Acta Neuropathologica, 1997, v. 93, n. 6, p. 628, doi. 10.1007/s004010050661
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- Article
Tripeptidyl Peptidase I, the Late Infantile Neuronal Ceroid Lipofuscinosis Gene Product, Initiates the Lysosomal Degradation of Subunit c of ATP Synthase1.
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- Journal of Biochemistry, 2000, v. 128, n. 3, p. 509, doi. 10.1093/oxfordjournals.jbchem.a022781
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- Article
The Intracellular Location and Function Of Proteins Of Neuronal Ceroid Lipofuscinoses.
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- Brain Pathology, 2004, v. 14, n. 1, p. 77, doi. 10.1111/j.1750-3639.2004.tb00501.x
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- Article
A novel gene expression scoring system for accurate diagnosis of basaloid squamous cell carcinoma of the esophagus.
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- International Journal of Oncology, 2017, v. 51, n. 3, p. 877, doi. 10.3892/ijo.2017.4075
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- Article
Differences in expression patterns of cathepsin C/dipeptidyl peptidase I in normal, pathological and aged mouse central nervous system.
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- European Journal of Neuroscience, 2013, v. 37, n. 5, p. 816, doi. 10.1111/ejn.12096
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- Article
Specific Delay of Degradation of Mitochondrial ATP Synthase Subunit c in Late Infantile Neuronal Ceroid Lipofuscinosis (Batten Disease).
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- Journal of Neurochemistry, 1995, v. 64, n. 2, p. 733, doi. 10.1046/j.1471-4159.1995.64020733.x
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- Article
Mutation of the glycosylated asparagine residue 286 in human CLN2 protein results in loss of enzymatic activity.
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- Glycobiology, 2004, v. 14, n. 4, p. 1, doi. 10.1093/glycob/cwh054
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- Article
Family with sequence similarity 83, member B is a predictor of poor prognosis and a potential therapeutic target for lung adenocarcinoma expressing wild-type epidermal growth factor receptor.
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- Oncology Letters, 2018, v. 15, n. 2, p. 1549, doi. 10.3892/ol.2017.7517
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- Article
Peroxisome degradation in mammals.
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- IUBMB Life, 2011, v. 63, n. 11, p. 1001, doi. 10.1002/iub.537
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Characterization of Cln3p, the gene product responsible for juvenile neuronal ceroid lipofuscinosis, as a lysosomal integral membrane glycoprotein.
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- Journal of Neurochemistry, 2003, v. 87, n. 5, p. 1296, doi. 10.1046/j.1471-4159.2003.02132.x
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- Article
A Lysosomal Proteinase, the Late Infantile Neuronal Ceroid LipofuscinosisGene (CLN2) Product, Is Essential for Degradation of a HydrophobicProtein, the Subunit c of ATP Synthase.
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- Journal of Neurochemistry, 1999, v. 72, n. 6, p. 2573, doi. 10.1046/j.1471-4159.1999.0722573.x
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Specific Delay in the Degradation of Mitochondrial ATP Synthase Subunit c in Late Infantile Neuronal Ceroid Lipofuscinosis Is Derived from Cellular Proteolytic Dysfunction Rather than Structural Alteration of Subunit c.
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- Journal of Neurochemistry, 1996, v. 67, n. 4, p. 1677, doi. 10.1046/j.1471-4159.1996.67041677.x
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- Article
Interleukin-11 Links Oxidative Stress and Compensatory Proliferation.
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- Science Signaling, 2012, v. 5, n. 207, p. 1, doi. 10.1126/scisignal.2002056
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- Article