Found: 7
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RasGAP-Derived Fragment N Increases the Resistance of Beta Cells towards Apoptosis in NOD Mice and Delays the Progression from Mild to Overt Diabetes.
- Published in:
- PLoS ONE, 2011, v. 6, n. 7, p. 1, doi. 10.1371/journal.pone.0022609
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- Publication type:
- Article
Genetic, cellular, and structural characterization of the membrane potential- dependent cell- penetrating peptide translocation pore.
- Published in:
- eLife, 2021, p. 1, doi. 10.7554/eLife.69832
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- Publication type:
- Article
APOBEC3C, a nucleolar protein induced by genotoxins, is excluded from DNA damage sites.
- Published in:
- FEBS Journal, 2022, v. 289, n. 3, p. 808, doi. 10.1111/febs.16202
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- Publication type:
- Article
Publisher Correction: Loss-of-function of the long non-coding RNA A830019P07Rik in mice does not affect insulin expression and secretion.
- Published in:
- 2020
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- Publication type:
- Correction Notice
Loss-of-function of the long non-coding RNA A830019P07Rik in mice does not affect insulin expression and secretion.
- Published in:
- Scientific Reports, 2020, v. 10, n. 1, p. 1, doi. 10.1038/s41598-020-62969-x
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- Publication type:
- Article
Expression of the NH(2)-terminal fragment of RasGAP in pancreatic beta-cells increases their resistance to stresses and protects mice from diabetes.
- Published in:
- 2009
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- Publication type:
- journal article
Expression of the NH<sub>2</sub>-Terminal Fragment of RasGAP in Pancreatic β-Cells Increases Their Resistance to Stresses and Protects Mice From Diabetes.
- Published in:
- Diabetes, 2009, v. 58, n. 11, p. 2596, doi. 10.2337/db09-0104
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- Publication type:
- Article