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Impact of Combined Baricitinib and FTI Treatment on Adipogenesis in Hutchinson–Gilford Progeria Syndrome and Other Lipodystrophic Laminopathies.
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- Cells (2073-4409), 2023, v. 12, n. 10, p. 1350, doi. 10.3390/cells12101350
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- Article
Impact of MnTBAP and Baricitinib Treatment on Hutchinson–Gilford Progeria Fibroblasts.
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- Pharmaceuticals (14248247), 2022, v. 15, n. 8, p. 945, doi. 10.3390/ph15080945
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- Article
Baricitinib, a JAK-STAT Inhibitor, Reduces the Cellular Toxicity of the Farnesyltransferase Inhibitor Lonafarnib in Progeria Cells.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 14, p. 7474, doi. 10.3390/ijms22147474
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- Article
Impact of Progerin Expression on Adipogenesis in Hutchinson—Gilford Progeria Skin-Derived Precursor Cells.
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- Cells (2073-4409), 2021, v. 10, n. 7, p. 1598, doi. 10.3390/cells10071598
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- Article
IL-17-Mediated Inflammation Promotes Cigarette Smoke-Induced Genomic Instability.
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- Cells (2073-4409), 2021, v. 10, n. 5, p. 1173, doi. 10.3390/cells10051173
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- Article
On Broken Ne(c)ks and Broken DNA: The Role of Human NEKs in the DNA Damage Response.
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- Cells (2073-4409), 2021, v. 10, n. 3, p. 507, doi. 10.3390/cells10030507
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- Article
Nuclear Pore Complexes Cluster in Dysmorphic Nuclei of Normal and Progeria Cells during Replicative Senescence.
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- Cells (2073-4409), 2021, v. 10, n. 1, p. 153, doi. 10.3390/cells10010153
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- Article
Inhibition of JAK-STAT Signaling with Baricitinib Reduces Inflammation and Improves Cellular Homeostasis in Progeria Cells.
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- Cells (2073-4409), 2019, v. 8, n. 10, p. 1276, doi. 10.3390/cells8101276
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- Article
Autophagic Removal of Farnesylated Carboxy-Terminal Lamin Peptides.
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- Cells (2073-4409), 2018, v. 7, n. 4, p. 33, doi. 10.3390/cells7040033
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- Article
Temsirolimus Partially Rescues the Hutchinson-Gilford Progeria Cellular Phenotype.
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- PLoS ONE, 2016, v. 11, n. 12, p. 1, doi. 10.1371/journal.pone.0168988
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- Article
Sulforaphane enhances progerin clearance in Hutchinson–Gilford progeria fibroblasts.
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- Aging Cell, 2015, v. 14, n. 1, p. 78, doi. 10.1111/acel.12300
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- Article
Induced Pluripotent Stem Cells Reveal Functional Differences Between Drugs Currently Investigated in Patients With Hutchinson-Gilford Progeria Syndrome.
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- Stem Cells Translational Medicine, 2014, v. 3, n. 4, p. 510, doi. 10.5966/sctm.2013-0168
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- Article
Defective Lamin A-Rb Signaling in Hutchinson-Gilford Progeria Syndrome and Reversal by Farnesyltransferase Inhibition.
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- PLoS ONE, 2010, v. 5, n. 6, p. 1, doi. 10.1371/journal.pone.0011132
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- Article
Epidermal expression of the truncated prelamin A causing Hutchinson–Gilford progeria syndrome: effects on keratinocytes, hair and skin.
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- Human Molecular Genetics, 2008, v. 17, n. 15, p. 2357, doi. 10.1093/hmg/ddn136
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- Article
Ligand-independent Regulation of the hairless Promoter by Vitamin D Receptor.
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- Photochemistry & Photobiology, 2008, v. 84, n. 2, p. 515, doi. 10.1111/j.1751-1097.2008.00301.x
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- Article
Dermal fibroblasts in Hutchinson-Gilford progeria syndrome with the lamin A G608G mutation have dysmorphic nuclei and are hypersensitive to heat stress.
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- BMC Cell Biology, 2005, v. 6, p. 27, doi. 10.1186/1471-2121-6-27
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- Article
Interaction of hairless and thyroid hormone receptor is not involved in the pathogenesis of atrichia with papular lesions.
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- Experimental Dermatology, 2004, v. 13, n. 4, p. 251, doi. 10.1111/j.0906-6705.2004.00174.x
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- Article
Mutation Report De novo mutations in monilethrix.
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- Experimental Dermatology, 2003, v. 12, n. 6, p. 882, doi. 10.1111/j.0906-6705.2003.00022.x
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- Article
Strategy to assess the efficiency of U1 RNA-hammerhead ribozyme constructs using GFP-tagged targets.
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- Experimental Dermatology, 2003, v. 12, n. 5, p. 712, doi. 10.1034/j.1600-0625.2003.00081.x
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- Article
Evidence for Extensive Locus Heterogeneity in Naxos Disease.
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- Journal of Investigative Dermatology, 2002, v. 118, n. 3, p. 557, doi. 10.1046/j.0022-202x.2001.01627.x
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- Article
Atrichia Caused by Mutations in the Vitamin D Receptor Gene is a Phenocopy of Generalized Atrichia Caused by Mutations in the Hairless Gene.
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- Journal of Investigative Dermatology, 2001, v. 117, n. 3, p. 612, doi. 10.1046/j.0022-202x.2001.01438.x
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- Article
Origin of the Beta Cells of the Islets of Langerhans is Further Questioned by the Expression of Neuronal Intermediate Filament Proteins, Peripherin and NF-L, in the Rat Insulinoma RIN5F Cell Line.
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- Developmental Neuroscience, 1991, v. 13, n. 6, p. 424, doi. 10.1159/000112194
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- Article